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Chickens exposed to loud noise can quickly regain hearing mostly due to repair of the tectorial membrane, not just hair cell regeneration.

Exosomes from umbilical cord cells fix hearing loss and damaged ear hair cells in mice.

Melatonin may protect inner ear cells from damage by reducing cell death and oxidative stress, potentially treating sudden hearing loss.

Melatonin may protect ear cells from damage caused by nicotine.

Deferoxamine may help protect inner ear cells from damage caused by oxidative stress.

Alopecia areata may be linked to hearing problems, so patients should monitor their hearing.

The new method makes it easier to study the whole cochlea from newborn mice and rats in the lab.

Follistatin and LIN28B together improve the ability of inner ear cells in mice to regenerate into hearing cells.

Caffeine can damage hearing cells and affect hearing recovery after ear trauma.

Estradiol may protect ear cells from hearing loss caused by a chemotherapy drug by activating a protective pathway.

4-octyl itaconate may prevent hearing loss caused by the drug cisplatin by activating certain cell protection pathways.

Blocking CISD1 reduces hearing loss from cisplatin in mice.

CaBP1 and CaBP2 are important for maintaining hearing by supporting continuous calcium currents and nerve signaling in the ear.

CaBP1 and CaBP2 are important for continuous hearing by preventing inactivation of calcium currents in ear cells, with CaBP2 also able to restore hearing when reintroduced.

A low dose of rapamycin increases inner ear hair cell creation by boosting SOX2+ cell numbers.

CaBP1 and 2 are necessary for maintaining calcium currents and hearing in inner ear cells.

The document concludes that there are no effective clinical treatments for hearing loss due to hair cell damage, but research is ongoing.

Clotrimazole, miconazole, and tolnaftate are likely safe, but gentian violet is risky.

Rapamycin reduces age-related hearing cell loss in mice, but acarbose does not.

Blocking autophagy increases survival of inner ear hair cells exposed to gentamicin.

Activin A speeds up ear hair cell differentiation, while Follistatin slows it down.

Higher minoxidil dose helps hair growth in non-responders without side effects.

Chemotherapy and radiation therapy cause skin and hair damage by altering gene expression and signaling pathways.

Early and late matrix progenitors in hair follicles create different cell layers, with early ones forming the companion layer and later ones forming the inner root sheath and hair shaft.

Sweat glands and hair follicles are determined by opposing signals, with BMPs promoting sweat glands and blocking BMPs leading to hair follicles.

Researchers improved a method to study individual cells in newborn mouse skin and found a way to assess the severity of a skin condition in humans.

The study found a link between the severity of Lichen Planopilaris seen by doctors and the details seen under a microscope, and created a new way to measure this severity.

Fibrinogen/LDL apheresis may improve speech perception in sudden hearing loss, especially for those with high fibrinogen and LDL levels.

Gene therapy with the Math1 gene helped regenerate balance-related cells and improve balance in mice.

Gene therapy, especially using atoh1, shows promise for creating functional sensory hair cells in the inner ear, but dosing and side effects need to be managed for clinical application.