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MOF controls key genes for skin development by regulating mitochondrial and ciliary functions.

Not having enough or having too much of the protein Grainyhead-like 3 leads to various developmental problems.

Gene therapy has potential as a future treatment for Hutchinson-Gilford progeria syndrome.

BMP2 and BMP7 have opposite roles in feather formation.

Certain genetic variations are linked to increased androgen levels in PCOS, but more research is needed to understand these connections fully.

TGF-β is crucial for tissue repair and can cause diseases if not properly regulated.

TRP channels in the skin are important for sensation and health, and targeting them could help treat skin disorders.

The WNT signaling pathway is important in many diseases and targeting it could offer new treatments.

Smart biomaterials that guide tissue repair are key for future medical treatments.

Some growth factors, while important for normal body functions, can cause diseases when not regulated properly.

Chicken feather gene mutation helps understand human hair disorders.

Tacrolimus causes fewer acute rejections than cyclosporin A in kidney transplants but doesn't necessarily improve kidney function after one year; cardiovascular risks and side effects vary between the two drugs.

Mouse skin can produce and process serotonin, with variations depending on hair cycle, body location, and mouse strain.

Men are more likely to get infectious skin diseases, while women are more prone to autoimmune and pigment-related skin conditions, influenced by biological and environmental factors.

Vitamin D receptor helps control hair growth genes in skin cells.

Skin patterns form through molecular signals and genetic factors, affecting healing and dermatology.

Pangolins have lost some skin-related genes, but kept others, leading to their unique scales and skin features.

Keratinocytes and adipose-derived stem cells can effectively heal difficult skin wounds.

More research is needed before using brown fat to treat polycystic ovary syndrome.

PCOS requires personalized treatment to improve life quality and reduce health risks.

The meeting highlighted the genetic basis of female pattern hair loss and various skin health insights.

The document summarizes medical findings on topics like heart rhythm treatment, sleep apnea therapy, and various health conditions and treatments.

Skin lesions in Carney Complex are caused by a gene change in some skin cells that leads to increased pigmentation and may lead to tumors.

Fat-derived stem cells show promise for skin repair and reducing aging signs but need more research for consistent results.

The document concludes that certain mutations may contribute to the inflammation in hidradenitis suppurativa and suggests that targeting TNFα could be a treatment strategy.

The study mapped yak skin cells to understand hair growth better.

The conclusion is that accurately replicating the complexity of the extracellular matrix in the lab is crucial for creating realistic human tissue models.

FGF9 controls which receptors it binds to through a process where two FGF9 molecules join, and changes in FGF9 can lead to incorrect receptor activation.

Lipocalin-Type Prostaglandin D2 Synthase (L-PGDS) is a protein that plays many roles in the body, including sleep regulation, pain management, food intake, and protection against harmful substances. It also affects fat metabolism, glucose intolerance, cell maturation, and is involved in various diseases like diabetes, cancer, and arthritis. It can influence sex organ development and embryonic cell differentiation, and its levels can be used as a diagnostic marker for certain conditions.

Removing Dicer from pigment cells in newborn mice causes early hair graying and changes in cell migration molecules.