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Intravenous calcium therapy is a safe and effective treatment for a rare type of rickets.

Intravenous and oral calcium effectively treated vitamin D dependent rickets type II, improving growth and bone health.

A genetic mutation caused severe rickets and alopecia in an Indian patient, but high-dose calcium and phosphate treatment improved their condition.

Tunisian children with hereditary vitamin D-resistant rickets showed improvement with calcium treatment, and new genetic mutations were identified.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

A 4-year-old boy with a rare type of rickets and hair loss improved in bone health but not hair growth after vitamin D and calcium treatment.

A child with rickets and hair loss might have a rare type of rickets that doesn't improve much with usual vitamin D and calcium treatment.

The enzyme Rand protease works well for leather dehairing and its stability is important, with Leu75 playing a key role.

A 3-year-old with vitamin D-resistant rickets and severe hair loss died despite treatment, highlighting the need for improved management of the condition.

Two rare genetic diseases cause severe rickets in children due to defects in vitamin D metabolism.

PRP therapy increases hair density for androgenetic alopecia.

Low-level laser therapy helps hair growth and reduces hair loss with few side effects.

Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

The created skin model with melanoblasts improves the study of skin color and offers an alternative to animal testing.

Involucrin gene expression is controlled by specific proteins and signaling pathways.

The nude gene causes skin cell overgrowth and improper development, leading to hair and urinary issues.

The R343H mutation in the VDR gene causes vitamin D-resistant rickets with alopecia by impairing specific gene activity.

MicroRNA-205 helps hair grow by changing the stiffness and contraction of hair follicle cells.

Testosterone affects androgen receptors and lipid storage in cells, while DHEA does not convert to testosterone or affect these receptors in the same way.

Mitochondrial reactive oxygen species are essential for skin and hair development.

Premature graying of hair is often linked with other health issues, but treatment with biotin, zinc, and calcium pantothenate is not very effective.

The research found two types of calcium in human hair, one that varies among individuals and another that is consistent across people.

The meeting discussed medical findings, including benefits of certain treatments for cancer and heart conditions, and highlighted issues like poor adherence to preventive measures and skill gaps among interns.

A man with hypoparathyroidism had other health issues that led to a diagnosis of a rare autoimmune disorder, APS-1.

Red light at 627 nm can safely trigger IL-4 release in skin cells, potentially helping treat inflammatory skin conditions.

Some drugs, especially biologics, can cause skin reactions that look like other skin diseases, and stopping the drug usually helps clear up these reactions.

An 11-year-old boy in Saudi Arabia has a rare case of hypoparathyroidism with severe brain calcifications but normal development and no known cause.

Hyaluronic acid and polycaprolactone improve skin regeneration, with polycaprolactone having a stronger effect on healing and tissue repair.

WWOX deficiency in mice causes skin and fat tissue problems due to disrupted cell survival signals.

Minoxidil, a blood pressure drug, can cause excessive hair growth, but this can be successfully treated with a hair removal agent called calcium thioglycolate.