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Hair and nail cells share similar proteins, indicating a common differentiation pathway.

Ectodin integrates BMP, SHH, and FGF signals in developing ectodermal organs.

Hair loss and aging are caused by the breakdown of a key protein in hair stem cells.

Androgens can both increase and decrease hair growth in different parts of the body.

A gene called APCDD1, which controls hair growth, is found to be faulty in a type of hair loss called hereditary hypotrichosis simplex.

Hair follicle dermal stem cells are key for regenerating parts of the hair follicle and determining hair type.

CDP is crucial for lung and hair follicle cell development.

Blocking JAK-STAT signaling can lead to hair growth.

Tight junctions are key for skin protection and controlling what gets absorbed or passes through the skin.

K6hf is a unique protein found only in a specific layer of hair follicles.

Macrophages help heal nerves by aiding the maturation of Schwann cells and are important for nerve repair.

The conclusion is that Treg-targeted therapies have potential, but more knowledge of Treg biology is needed for effective treatments, including for cancer.

Stem cell plasticity is crucial for wound healing but can also contribute to cancer development.

The AUTS2 gene is linked to neurological disorders and may affect human brain development and cognition.

MicroRNA-214 is important for skin and hair growth because it affects the Wnt pathway.

Hormones significantly affect hair and oil gland function in the skin, and more research is needed on skin-related hormone disorders.

The p75 neurotrophin receptor is important for hair follicle regression by controlling cell death.

The Wnt/β-catenin pathway helps tissue regeneration but can also cause fibrosis, and drugs that inhibit this pathway may aid in healing skin and heart tissues.

A new mutation in the human glucocorticoid receptor reduces its function and causes resistance to glucocorticoids.

The Foxn1 gene mutation causes hairlessness and immune system issues, and understanding it could lead to hair growth disorder treatments.

Animal models have helped understand hair loss from alopecia areata and find new treatments.

Lichen Planopilaris and Frontal Fibrosing Alopecia may help us understand hair follicle stem cell disorders and suggest new treatments.

Mutations in Gasdermin A3 cause skin inflammation and hair loss by disrupting mitochondria.

Prolactin affects when mice shed and grow hair.

Cancer can hijack the body's cell repair system to promote tumor growth, and targeting this process may improve cancer treatments.

Tissue-resident memory T cells can protect against infections and cancer but may also contribute to autoimmune diseases.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

Inactive hair follicle stem cells help prevent skin cancer.

Kennedy's disease leads to muscle weakness and sensory issues, has no cure but manageable symptoms, and future treatments look promising.

A deficiency in the TTC7A gene causes immune problems, gut issues, and hair loss.