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Balding hair follicle cells are smaller, grow less well, and need more effort to culture than non-balding cells.

Cells from balding scalps have more androgen receptors than cells from non-balding scalps.

Hair from balding and non-balding areas regrows similarly on mice.

SFRP2 and PTGDS may be key factors in female hair loss.

Problems with arginine processing in hair follicles might contribute to common hair loss, and arginine supplements could help treat it.

A substance called DKK-1 increases in balding areas and causes hair cells to die when exposed to DHT.

Changing hair follicle identity could potentially reverse balding.

The gene LRRC15 is more active in balding areas of the scalp compared to non-balding areas.

Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

Balding men had lower bone density, while balding women had higher hip bone density.

Aging scalp skin contributes to hair aging and loss, and more research is needed to develop better hair loss treatments.

Increased cell death and reduced cell growth in hair follicles contribute to baldness.

Hormones and their receptors, especially androgens, play a key role in hair growth and disorders like baldness.

Wnt/β-catenin and TGF-β pathways affect hair loss, and activating Wnt/β-catenin could be a potential treatment.

High testosterone to epitestosterone ratio in hair could predict male-pattern baldness.

miR-122 causes hair loss by killing hair cells.

Some children may develop hair loss after having COVID-19.

IGF-1 affects hair loss and could be a potential treatment.

Androgenetic alopecia, or hair loss, is caused by a mix of genetics, hormones, and environment, where testosterone affects hair growth and causes hair to become smaller and grow for a shorter time.

Androgen receptor signaling causes early aging of cells important for hair growth by damaging their DNA.

Men with hair loss have more DNA changes in back-of-head hair follicles, possibly protecting them from thinning.

Male hormones cause different growth in identical human hair follicles due to their unique epigenetic characteristics.

Hair follicle biology advancements may lead to better hair growth disorder treatments.

Finasteride helps hair growth but stopping it causes faster hair loss.

Androgens may cause hair loss by increasing TGF-beta1 from scalp cells, which inhibits hair cell growth.

Male hormones and their receptors play a key role in hair loss and skin health, with potential new treatments being explored.

The osteopontin gene is active in a specific part of rat hair follicles during a certain hair growth phase and might affect hair cycle and diseases.

Hair loss in Androgenetic Alopecia is caused by genetics, aging, and lifestyle, leading to hair follicle shrinkage and related health risks.

Baldness might be caused by scalp weight pressing on hair follicles.