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Hair sensitivity to androgens is partly controlled by specific enzyme expressions in different hair areas.

Androgens are important for normal ovarian function and estrogen production, but may not be the main cause of follicle death.

The balance between androgen receptor and p53 is crucial for sebaceous gland differentiation.

AR polyglycine repeat doesn't cause baldness.

Testosterone replacement can help women with low libido and mood, but they need to have enough estrogen first to avoid side effects.

Prostate cells have proteins that bind to specific hormones, which can increase protein production when activated by these hormones.

Different types of androgens bind differently to two receptors, AR1 and AR2, in Atlantic croaker's brain and ovarian tissues, suggesting these receptors may control different androgen actions in fish.

Steroidogenesis inhibitors change but don't stop androgen production in prostate cancer.

Androgens increase a growth factor in hair cells by creating reactive oxygen species, and antioxidants might help treat hair loss.

Two enzymes regulate androgen receptor activity, affecting treatments for androgen insufficiency and benign prostatic hyperplasia.

Hair loss in women may have causes other than hormones.

The length of the CAG repeat in the androgen receptor gene affects the risk and progression of prostate cancer, BPH, infertility, and undermasculinized genitalia.

Androgen conjugates might be better indicators of skin sensitivity to hormones in women with excessive hair growth.

Reduced AR gene methylation may cause early pubic hair growth in girls.

The research found that anovulatory young women have higher androgen and hormone levels than those who ovulate, suggesting immature hormonal regulation rather than a specific condition.

Beard cells, unlike scalp cells, produce growth factors in response to testosterone, which may explain differences in hair growth.

Men with male-pattern baldness have more androgen receptors in their scalp's oil glands, which may contribute to hair loss.

Low androgen levels might delay prostate cancer but could lead to more aggressive, therapy-resistant cancers.

Hidradenitis suppurativa improves with antiandrogen therapy.

Use "female pattern hair loss" term, assess androgen excess, treat with minoxidil and other medications if needed.

Testosterone helps beard and axillary hair cells grow by releasing growth factors from dermal papilla cells.

Dietary changes, including weight loss and a balanced diet, are important for managing PCOS, especially in overweight women.

Ludwig pattern hair loss in women results from varying sensitivity in hair follicles, causing fewer visible hairs.

Genetic defects in androgen production are linked to male developmental disorders and are improving treatment understanding.

The conclusion is that androgen receptor gene polymorphism might be a marker for polycystic ovary syndrome, but more research is needed.

Too much AKR1C3 enzyme causes resistance to finasteride by increasing testosterone.

New compounds with carborane showed anti-androgen effects similar to flutamide.

Longer CAG repeat lengths in the androgen receptor gene are linked to higher testosterone levels in women with PCOS.

The document concludes that the androgen receptor's structure and function are complex, affecting how it regulates genes and is involved in diseases like prostate cancer.

Hormonal male contraception is effective and could be market-ready, but more research is needed to ensure safety for all ethnic groups.