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Certain genetic markers linked to reproductive potential were identified by their impact on a protein's ability to bind to genes.

Chinese domestic goats have unique genetic traits due to domestication and geographic isolation.

Cellular senescence has both cancer-blocking and cancer-promoting effects, and targeting senescent cells may improve health and lifespan.

A new mutation in the STING protein causes a range of symptoms and its severity may be affected by other genetic variations; treatment with a specific inhibitor showed improvement in one patient.

Genetic variants linked to ten skin diseases were found, showing both immune and non-immune factors play a role.

Blocking β-catenin in skin cells improves hair growth during wound healing.

Using SNP array testing helped quickly find the gene causing Woodhouse-Sakati syndrome in two related individuals.

Protein profiling of forehead skin can help distinguish between frontal fibrosing alopecia and androgenetic alopecia.

CYLD mutations cause a variety of skin tumors with symptoms starting around age 16, and treatments are currently limited.

Genetic variants in PDE6H and LPAR6 cause vision impairment and hair loss in two Pakistani brothers.

Meis1 is crucial for skin health and tumor development.

Alopecia areata shows a unique type 1 interferon signature, suggesting potential treatment by targeting this pathway.

Neuregulin3 affects cell development in the skin and mammary glands.

Blocking Wnt signaling in young mice causes thymus shrinkage and cell loss, but recovery is possible when the block is removed.

The NUDT15 gene variant causes severe side effects from azathioprine in some Indian patients.

Common variants in the Trichohyalin gene are linked to straight hair in Europeans.

Skin lesions in Carney Complex are caused by a gene change in some skin cells that leads to increased pigmentation and may lead to tumors.

A mother's PPARγ is crucial for preventing harmful milk that can cause inflammation and growth problems in babies.

The 2015 Hair Research Congress concluded that stem cells, maraviroc, and simvastatin could potentially treat Alopecia Areata, topical minoxidil, finasteride, and steroids could treat Frontal Fibrosing Alopecia, and PTGDR2 antagonists could also treat alopecia. They also found that low-level light therapy could help with hair loss, a robotic device could assist in hair extraction, and nutrition could aid hair growth. They suggested that Alopecia Areata is an inflammatory disorder, not a single disease, indicating a need for personalized treatments.

The document summarizes medical findings on topics like heart rhythm treatment, sleep apnea therapy, and various health conditions and treatments.

The document concludes that early diagnosis and treatment of Congenital Adrenal Hyperplasia are crucial for preventing serious health issues and improving patient outcomes.

The meeting highlighted the genetic basis of female pattern hair loss and various skin health insights.

The document concludes that accurate diagnosis of different types of hair loss requires careful examination of hair and scalp tissue, considering both clinical and microscopic features.

CCC1 is essential for ion balance and proper plant cell function.

CCC1 is essential for pH balance and normal cell function in plants.

Prdm1 is necessary for early whisker development in mice but not for other hair, and its absence changes nerve and brain patterns related to whiskers.

Low serum complement levels in SLE patients don't always match with disease flares; monitoring C3 and C4 is useful, but cell-bound complement products might better indicate disease activity.

Alopecia areata causes hair loss due to an immune attack on hair follicles, influenced by genetics and environment.

Some drugs can cause skin, nail, and hair problems, which are important for healthcare professionals to recognize and report.