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Mutations in the p63 gene affect skin adhesion, barrier integrity, and hair growth.

Controlling Tslp can improve health in AEC syndrome patients.

Early and aggressive treatment is crucial for preserving vision in infants with AEC syndrome.

Rapp-Hodgkin syndrome, AEC, and EEC are different expressions of the same genetic disorder caused by TP63 gene mutations.

The commentary stresses the importance of using historical control data and proper interpretation in evaluating developmental toxicity.

CD10 and CD34 levels change during hair development and different hair growth stages, which could be important for hair regeneration treatments.

The Wnt/β-catenin pathway helps tissue regeneration but can also cause fibrosis, and drugs that inhibit this pathway may aid in healing skin and heart tissues.

Mice that can regenerate tissue have cells that pause in the cell cycle, which is important for healing, similar to axolotls.

Strong skin creams work well for long-term scalp inflammation in Rapp-Hodgkin Ectodermal Dysplasia.

Apoptosis is essential for human skin development and forming a functional epidermis.

Melatonin in the skin helps protect against damage from stress and UV rays, and could be used to treat certain skin conditions.

Lactate production is important for activating hair growth stem cells.

Regulatory T-cells are important for healing and regenerating tissues in various organs by controlling immune responses and aiding stem cells.

Cathepsin L is essential for normal hair growth and development.

Male hormones affect COVID-19 severity and certain drugs targeting these hormones could help reduce the risk.

Damage to skin releases dsRNA, which activates TLR3 and helps in skin and hair follicle regeneration.

Prolactin contributes to hair loss by promoting hair follicle shrinkage and cell death.

Fibroblast growth factor receptor signaling controls cell development and repair, and its malfunction can cause disorders and cancer, but it also offers potential for targeted therapies.

Hair grows faster in the morning and is more vulnerable to damage from radiation due to the internal clock in hair follicle cells.

Hair, teeth, and mammary glands develop similarly at first but use different genes later.

A new gene mutation causes female pseudohermaphroditism due to glucocorticoid resistance.

Prolactin affects hair growth cycles and can cause early hair follicle regression.

The Wnt/β-catenin pathway is important for tissue development and has potential in regenerative medicine, but requires more research for therapeutic use.

Mammals might fail to regenerate not because they lack the right cells, but because of how cells respond to their surroundings, and changing this environment could enhance regeneration.

Minoxidil boosts hair growth by activating PGHS-1.

Smart biomaterials that guide tissue repair are key for future medical treatments.

Treatment with plasma rich in growth factors improved hair density and thickness for hair loss patients.

Advanced human skin models improve drug development and could replace animal testing.

Prostaglandin D2 blocks new hair growth after skin injury through the Gpr44 receptor.

Inactive hair follicle stem cells help prevent skin cancer.