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XEDAR triggers a specific signaling pathway in cells.

EdnrB signaling helps melanocyte stem cells regenerate and could be targeted to treat pigmentation issues.

Progesterone and its byproducts control a specific receptor in the brain independently of progesterone receptors, affecting conditions related to the menstrual cycle.

Mice lacking fibromodulin have disrupted healing patterns, leading to abnormal skin repair and scarring.

Pregnant women with PCOS have higher levels of Neurokinin B in the placenta, especially with female babies.

The document explains the difficulty in diagnosing and treating brain diseases caused by the immune system and stresses the need for quick and accurate tests.

The conclusion is that the nuclear lamina and LINC complex in skin cells respond to mechanical signals, affecting gene expression and cell differentiation, which is important for skin health and can impact skin diseases.

A specific signal from hair cells controls the tightening of the surrounding muscle, which is necessary for hair shedding.

Eph/ephrin signaling is important for skin cell behavior and could be targeted to treat skin diseases.

SDF-1/CXCL12 and its receptor CXCR4 are crucial for melanocyte movement in mouse hair follicles.

Too much Smad7 can cause serious changes in skin tissues, including problems with hair growth, thymus shrinkage, and eye development issues.

Terbinafine can cause hair loss.

Neuroactive steroids could become safe, effective treatments with more understanding of their complex brain actions and metabolism.

Fibromodulin might help reduce scarring if increased in adult wounds like in fetal skin that heals without scars.

Dendrobium officinale polysaccharides may help with hair growth, skin moisturization, and protection against oxidative damage.

The update highlights that non-classic congenital adrenal hyperplasia is common in women with excess male hormones, requires specific hormone tests for diagnosis, and has various treatment options depending on age and symptoms.

Skin lesions in Carney Complex are caused by a gene change in some skin cells that leads to increased pigmentation and may lead to tumors.

Too much Smad7 changes skin and hair development by breaking down a protein called β-catenin, leading to more oil glands and fewer hair follicles.

Wnt/β-catenin and TGF-β pathways affect hair loss, and activating Wnt/β-catenin could be a potential treatment.

Skin lesions in Carney complex are likely caused by a specific group of skin cells that promote pigment production due to a genetic mutation.

The document concludes that new treatments are needed to better manage acne and reduce side effects related to current therapies.

Antiandrogens have important biological effects, but more research is needed to understand them fully and compare their effectiveness and side effects to other treatments.

Helminth protein helps wounds heal better by reducing scarring and promoting tissue growth.

Oral cyclosporin doesn't stop hair loss.

Blocking JAK-STAT signaling can lead to hair growth.

Genes linked to fibrosis are more active in people with central centrifugal cicatricial alopecia.

Neurotoxins can affect neurotransmitter release and have potential in treating muscle, pain, and cancer conditions, but more research is needed on how they work.

The nucleus is key in controlling skin growth and repair by coordinating signals, gene regulators, and epigenetic changes.

The TGF-β family helps control how cells change and move, affecting skin, hair, and organ development.

Integrin alphavbeta6 is important for wound healing and hair growth, and blocking it may improve these processes.