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Minoxidil boosts hair growth by undergoing a process in hair follicles and certain skin cells, and by increasing the production of compounds essential for hair growth and maintenance.

The conclusion is that tritium-labeled testosterone metabolites can be made and are better converted into dihydrotestosterone in skin cells than in prostate tissue.

Sebocytes play a key role in controlling androgen levels in human skin.

Finasteride effectively inhibits 5α reductase, while plant extracts like Permixon and Bazoton don't show significant results.

EGF makes hair follicles grow longer but stops hair production.

Men with male-pattern baldness have more androgen receptors in their scalp's oil glands, which may contribute to hair loss.

Topical finasteride and flutamide reduce gland size and enzyme activity, with flutamide being more potent, potentially treating acne, seborrhea, hirsutism, and androgenic alopecia.

bFGF, VEGF, and minoxidil decrease collagen production in hair cells, possibly affecting hair growth.

OCT binds strongly to hair sheath cells and may affect skin and hair growth with fewer side effects than vitamin D3.

Certain sex hormones and antiandrogens can either slow down or speed up the growth of human hair follicle cells depending on their concentration.

Rat skin takes up and processes testosterone differently than other organs, with testosterone being more important for the skin than its metabolite 5α-DHT.

Minoxidil absorption in skin is slowed by cleansing, depends on how long it stays on the skin, and is not much affected by reapplication.

Melanocyte-associated antigens may play a key role in alopecia areata and could be targets for new treatments.

The Ayurvedic treatment helped a woman with PCOS, improving her symptoms and menstrual cycle.

People with hair loss have more androgen receptors and enzymes in certain follicles, with men and women showing different patterns.

Finasteride effectively blocks enzyme causing male pattern baldness.

Liposomes and niosomes improve finasteride delivery for hair loss treatment.

Minoxidil promotes hair growth but stops working when discontinued.

Liposomes can safely and effectively deliver substances to mouse hair follicles, potentially useful for human hair treatments.

Dihydrotestosterone increases the activity of an enzyme in pubic skin cells that converts testosterone to dihydrotestosterone.

Androgens like testosterone affect hair growth and oil production differently across body parts and individuals.

The effects of estrogen on human hair growth are unclear and need more research.

New steroidal compounds could be effective for treating conditions related to 5α-reductase enzyme activity.

Human platelets change minoxidil to minoxidil sulfate, helping blood vessels widen.

These drugs raise prostaglandin-like material in dog blood, possibly causing blood vessel widening.

The conclusion is that small hair follicles cause baldness in macaques, and treatments like antiandrogens and minoxidil can prevent hair loss and promote regrowth.

Some stem cells in the body rarely divide, which could help create better treatments for diseases and aging.

Finasteride potentially treats hair loss by reducing DHT production.

Minoxidil reduces lysine hydroxylase in skin cells.

A cream with a specific hormone blocker increases hair growth in mice.