Search

everythinglearnresearchcommunity

for

Search:↓

The study found that certain mutations in the vitamin D receptor can cause rickets and potentially affect hair growth.

Vitamin D receptor binds similarly to natural and synthetic ligands, affecting gene regulation.

Vitamin D receptor interacts with certain dietary components to help prevent diseases and regulate hair growth.

The vitamin D receptor can work without its usual activating molecule.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

LEF1 interacts with Vitamin D Receptor, affecting hair follicle regeneration and this could be linked to hair loss conditions.

Two rare genetic diseases cause severe rickets in children due to defects in vitamin D metabolism.

NcoA4 may have roles beyond helping control gene activity, possibly affecting cell behavior and stability.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

The R343H mutation in the VDR gene causes vitamin D-resistant rickets with alopecia by impairing specific gene activity.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

Most Hairless gene mutations reduce its ability to work with the Vitamin D Receptor, which might explain a certain type of hair loss.

Hairless protein is crucial for healthy skin and hair, and its malfunction can cause hair loss.

Hormones and their receptors, especially androgens, play a key role in hair growth and disorders like baldness.

The vitamin D receptor helps maintain hair and bone health even without binding vitamin D.

Lampreys have a functional vitamin D receptor that may help detoxify harmful substances.

Researchers created a mouse model of a type of rickets that does not cause hair loss.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

Mice without the vitamin D receptor have bone issues and other health problems, suggesting vitamin D is important for preventing various diseases in humans.

Vitamin D has potential benefits for cancer prevention, heart health, diabetes, obesity, muscle function, skin health, and immune function, but clinical results are mixed and more research is needed.

The vitamin D receptor is crucial for bone health and affects various body systems, with mutations potentially leading to disease.

The review found no clear link between vitamin D receptor gene variations and polycystic ovary syndrome.

Vitamin D receptor helps control hair growth genes in skin cells.

The Hairless gene is crucial for healthy skin and hair growth.

Gene therapy helped rats with a specific type of rickets grow hair without severe inflammation.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

The vitamin D receptor helps regulate skin and hair health independently of its usual vitamin D ligand.

Vitamin D receptor is crucial for skin wound healing.

Vitamin D is important for bones, hair, blood pressure, and breast development.