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The guide explains how to study human and mouse sebaceous glands using various staining and imaging techniques, and emphasizes the need for standardized assessment methods.

Edar signaling is crucial for controlling hair growth and regression.

Different melanocyte types in hair follicles either survive or die during the catagen phase.

Stress in mice delays hair growth and treatments blocking substance P can partly reverse this effect.

Aging and sun damage do not increase the skin's absorption of certain sunscreens and drugs.

TGF-β2 helps yak hair follicles enter the regression phase, while HSP70 tries to prevent it.

Apoptosis helps shape hair growth and prepares the skin for hair to emerge.

Melatonin reduced oxidative damage, inflammation, and protected hair cells in scalded rats.

Apoptosis contributes to hair loss in androgenetic alopecia.

HIV-1 may cause increased stem cell death in hair follicles, leading to hair loss.

Testosterone can both promote hair growth and cause baldness by affecting hair growth signals.

Tetrahydroxystilbene Glucoside may help prevent hair loss by blocking certain pathways that lead to cell death.

Apoptosis may contribute to hair loss in androgenetic alopecia.

Tofacitinib was more effective than apremilast in treating hair loss in a mouse model of alopecia areata.

Innate lymphoid cells type 1 may contribute to alopecia areata by damaging hair follicles.

PRP helps hair regrowth and thickness.

Apoptosis is essential for human skin development and forming a functional epidermis.

Stress stops hair growth in mice by causing early hair growth phase end and harmful inflammation through a specific nerve-related pathway.

Understanding hair growth involves complex interactions between molecules and could help treat hair disorders.

Cathepsin L is essential for normal hair growth and development.

Prolactin contributes to hair loss by promoting hair follicle shrinkage and cell death.

Stress can cause hair loss by negatively affecting hair follicles and this effect might be reversed with specific treatments.

The document concludes that assessing hair follicle damage due to cyclophosphamide in mice involves analyzing structural changes and suggests a scoring system for standardized evaluation.

All-trans retinoic acid causes hair loss by increasing TGF-β2 in hair follicle cells.

Mutant mice help researchers understand hair growth and related genetic factors.

Mice without the Sept4/ARTS gene heal wounds better due to more stem cells that don't die easily.

Hair greying is caused by oxidative stress damaging hair follicles and melanocytes.

The document concludes that there are no effective clinical treatments for hearing loss due to hair cell damage, but research is ongoing.

Noggin overexpression delays eyelid opening by affecting cell death and skin cell development.

PEGL-DOX causes Hand-Foot Syndrome due to skin reactions from prolonged circulation and ROS generation.