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Mice with extra human KLK14 had hair and skin problems, including weaker cell bonds and inflammation, linked to Netherton syndrome.

SV40 T antigen in hair follicles causes abnormal hair and health issues in mice.

Overexpressing SSAT in mice makes them highly sensitive to polyamine analogues, causing liver damage and high mortality.

The HPV type 11 region activates hair-specific gene expression in mice.

Proteolytic enzymes damage hair follicles by detaching stem cells.

Mice with human chymase had a higher death rate when exposed to a toxin compared to normal mice.

Scientists made a mouse that can be made to lose hair and then grow it back.

Overexpressing ovine β-catenin in mice skin increases hair follicle density and growth.

Proteolytic enzymes damaged hair follicle stem cells in transgenic mice.

Mice with a changed Hr gene lose and regrow hair due to changes in the gene's activity.

The TCL1 transgenic mouse model is useful for understanding human B-cell leukemia and testing new treatments.

GFP transgenic mice help study cell origins in skin grafts.

Overexpressing follistatin in mice delays wound healing and reduces scar size.

Overexpressing a specific enzyme in mice causes hair loss and female infertility.

K6/ODC transgenic mice are effective for quickly identifying cancer-causing chemicals.

Excessive putrescine causes hair loss in transgenic mice by disrupting hair follicle development.

Overexpression of SSAT causes hair loss and skin issues, but reducing putrescine can help.

IGF-1 increases whisker growth in transgenic mice.

Adding TERT and BMI1 to certain skin cells can improve their ability to create hair follicles in mice.

Erythropoietin overexpression disrupts hair growth and fat formation in mice.

The system allows precise control of gene expression in mouse skin, useful for studying skin biology.

Overexpression of activin A in mice skin causes skin thickening, fibrosis, and improved wound healing.

Mice with human skin protein K8 had more skin problems and cancer.

Increasing COX-2 in mouse skin causes bigger sebaceous glands and thinner hair, but stopping COX-2 can reverse hair thinning.

Researchers created a new mouse model, G4, that mimics human PCOS symptoms and links the condition to a specific gene.

Overexpressing thrombospondin-1 in mice skin prevents UVB-induced skin damage.

Alpha-difluoromethylornithine prevents cancer in mice but causes hair loss.

Deleting TNFα gene reduces skin cancer risk in certain mice.

High levels of ODC and a mutant Ha-ras gene cause tumors in mice.