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Research 30 of 723
- Defining the impact of β-catenin/Tcf transactivation on epithelial stem cells
- TCF/Lef1-Mediated Control of Lipid Metabolism Regulates Skin Barrier Function
- Miz1 is required for hair follicle structure and hair morphogenesis
- MAD2B acts as a negative regulatory partner of TCF4 on proliferation in human dermal papilla cells
- In vivo transcriptional governance of hair follicle stem cells by canonical Wnt regulators
- Androgen modulation of Wnt/β-catenin signaling in androgenetic alopecia
- Hairless is a nuclear receptor corepressor essential for skin function
- Tcf3 and Lef1 regulate lineage differentiation of multipotent stem cells in skin
- Targeting the Wnt pathways for therapies
- Data from Activator Protein-1 Activity Regulates Epithelial Tumor Cell Identity
- 皮膚組織の再生に向けた皮膚由来幹細胞に関する研究
- ROR2 regulates self-renewal and maintenance of hair follicle stem cells
- Epidermal homeostasis: a balancing act of stem cells in the skin
- Scratching the surface of skin development
- The hair cycle
- β-catenin Activity in the Dermal Papilla Regulates Morphogenesis and Regeneration of Hair
- Mesenchymal–epithelial interactions during hair follicle morphogenesis and cycling
- Wnt Signaling in Skin Development, Homeostasis, and Disease
- Coordinated Activation of Wnt in Epithelial and Melanocyte Stem Cells Initiates Pigmented Hair Regeneration
- Signaling Involved in Hair Follicle Morphogenesis and Development
- More than one way to skin . . .
- Stem cell niches in mammals
- Macrophages Contribute to the Cyclic Activation of Adult Hair Follicle Stem Cells
- Mechanical stretch induces hair regeneration through the alternative activation of macrophages
- Smad7-Induced β-Catenin Degradation Alters Epidermal Appendage Development
- Development and homeostasis of the sebaceous gland
- The hair cycle
- Regenerative Hair Waves in Aging Mice and Extra-Follicular Modulators Follistatin, Dkk1, and Sfrp4
- Hairless and Wnt Signaling: Allies in Epithelial Stem Cell Differentiation
- mTOR signaling promotes stem cell activation via counterbalancing BMP-mediated suppression during hair regeneration