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Lack of Ctip2 in skin cells delays wound healing and disrupts hair follicle stem cell markers in mice.

Skin stem cells interacting with their environment is crucial for maintaining and regenerating skin and hair, and understanding this can help develop new treatments for skin and hair disorders.

TGF-β2 helps activate hair follicle stem cells by counteracting BMP signals.

Inactive stem cells in hair follicles and muscles can avoid detection by the immune system.

Prdm1 is necessary for early whisker development in mice but not for other hair, and its absence changes nerve and brain patterns related to whiskers.

Hair and skin can regenerate without bulge stem cells due to other compensating cells.

Wounded skin cells can revert to stem cells and help heal.

Vitamin D helps skin stem cells heal wounds by working with a key skin protein.

Blocking JAK-STAT5 signaling in mice leads to hair growth.

The research suggests new treatments for skin cancer could target specific cell growth pathways.

Chemotherapy and radiotherapy can cause skin side effects like rashes, hair loss, and nail changes, which are usually managed with conservative treatments.

Claudin-1 is crucial for maintaining skin barrier and preventing inflammation.

Lack of Crif1 in hair follicle stem cells slows down hair growth in mice.

Hair growth-related cells need the enzyme SCD1 to help maintain the area that supports hair growth.

The new CoQ10 gel protects mouse skin better against aging from UV light than the old gel.

Cooling the scalp may prevent hair loss from chemotherapy, hair often grows back after treatment, and nail issues usually improve after stopping the drug.

Current treatments for hair loss from chemotherapy are limited, but new methods are being researched.

Some chemicals and drugs can cause hair loss, which usually grows back after stopping the treatment.

Nerve signals are crucial for hair follicle stem cells to become skin stem cells and help in wound healing.

Epidermal stem cells show promise for future dermatology treatments due to ongoing advancements.

AP-2α and AP-2β proteins are essential for healthy adult skin and hair.

Skin needs dermal β-catenin activity for hair growth and skin cell multiplication.

Sweat gland development involves two unique skin cell programs and a temporary skin environment.

Jagged-1 in skin Tregs is crucial for timely wound healing by recruiting specific immune cells.

Estrogens significantly influence hair growth by interacting with receptors in hair follicles and may help regulate the hair growth cycle.

A protein called IL-36γ causes skin side effects from certain cancer treatments when combined with a common skin bacteria.

Skin cancer often starts from Lgr5+ progenitor cells.

GDNF signaling helps in hair growth and skin healing after a wound.

Some drugs can cause changes to your hair.

The document concludes that alopecia has significant social and psychological effects, leading to a market for hair loss treatments.