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Prolactin affects hair growth and skin conditions, and could be a target for new skin disease treatments.

The nucleus is key in controlling skin growth and repair by coordinating signals, gene regulators, and epigenetic changes.

CRH in the skin acts like the body's stress response system, affecting cell behavior and immune activity.

Hairless protein is crucial for healthy skin and hair, and its malfunction can cause hair loss.

Immune cells affect hair growth and could lead to new hair loss treatments.

Human foreskin does not show aging or reduced cell growth after radiation, and H2A.J is not a good marker for radiation-induced aging.

Sweat gland development involves two unique skin cell programs and a temporary skin environment.

Chemotherapy and radiation therapy cause skin and hair damage by altering gene expression and signaling pathways.

Fat-derived stem cells may help treat skin aging and hair loss.

Skin needs dermal β-catenin activity for hair growth and skin cell multiplication.

Dihydrotestosterone (DHT) doesn't affect rat skin cell growth, but it does change cell cycle, protein levels, and other cell functions, potentially shortening hair growth cycle.

Skin has specialized touch receptors that can tell different sensations apart.

Mesenchymal stem cells help improve wound healing by reducing inflammation and promoting skin cell growth and movement.

Merkel cells stabilize nerve endings in the skin, and they change independently of each other.

Bone Morphogenetic Proteins (BMPs) help control skin health, hair growth, and color, and could potentially be used to treat skin and hair disorders.

TRP channels in the skin are important for sensation and health, and targeting them could help treat skin disorders.

Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

Mesenchymal stem cells show potential for skin healing and anti-aging, but more research is needed for safe use, especially regarding stem cells from induced pluripotent sources.

Gene network oscillations inside hair stem cells are key for hair growth regulation and could help treat hair loss.

Nuclear shape and chromatin changes affect gene expression in skin cell differentiation.

Bioprinting could improve wound healing but needs more development to match real skin.

The new skin patch with human matrix and antibiotic improves wound healing.

Researchers created early-stage hair-like structures from skin cells, showing how these cells can self-organize, but more is needed for complete hair growth.

3D bioprinting could improve skin repair and treat conditions like vitiligo and alopecia by precisely placing cells.

Nestin-expressing cells turn into a specific type of skin cell in hair follicles during development and in adults.

Mouse skin can produce and process serotonin, with variations depending on hair cycle, body location, and mouse strain.

Nanoparticles can enter the skin, potentially causing toxicity, especially in damaged skin.

The study concluded that hair growth in mice is regulated by a stable interaction between skin cell types, and disrupting this can cause hair loss.

Fat-derived stem cells and their secretions show promise for treating skin aging and hair loss.

Androgen receptor activity blocks Wnt/β-catenin signaling, affecting hair growth and skin cell balance.