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Different signals work together to change gene activity and guide hair follicle stem cells to become specific cell types.

Tet1/2/3 enzymes affect hair follicle cell development by influencing BMP signaling.

Smad1 and Smad5 are essential for hair follicle development and stem cell sleepiness.

Mouse skin glands need healthy nerves to grow properly during hair growth phases.

Hair follicles repair 3D injuries using a 2D healing process.

African spiny mice can regenerate skin, hair, and cartilage, but not muscle, and their unique abilities could be useful for regenerative medicine.

Some wound-healing cells can turn into fat cells around new hair growth in mice.

TGF-β is crucial for tissue repair and can cause diseases if not properly regulated.

TGF-β is crucial for controlling stem cell behavior and changes in its signaling can lead to diseases like cancer.

Too much Smad7 changes skin and hair development by breaking down a protein called β-catenin, leading to more oil glands and fewer hair follicles.

Stem cell plasticity is crucial for wound healing but can also contribute to cancer development.

The document concludes that while significant progress has been made in understanding skin biology and stem cells, more research is needed to fully understand their interactions with their environment.

MicroRNA-214 is important for skin and hair growth because it affects the Wnt pathway.

The TGF-β family helps control how cells change and move, affecting skin, hair, and organ development.

mTOR signaling helps activate hair stem cells by balancing out the suppression caused by BMP during hair growth.

Inactive hair follicle stem cells help prevent skin cancer.

Noggin overexpression delays eyelid opening by affecting cell death and skin cell development.

The study showed that hair follicle stem cells can maintain and organize themselves in a lab setting, keeping their ability to renew and form hair and skin.

Hair can regrow after certain stem cells are lost because other stem cells can take over their role.

Wnt, Eda, and Shh pathways are crucial for different stages of sweat gland development in mice.

Fat cells are important for tissue repair and stem cell support in various body parts.

Blocking a specific protein signal can make hair grow on mouse nipples.

BMP6 and Wnt10b control whether hair follicles are resting or growing.

The protein SLC1A3 is important for activating skin stem cells and is necessary for normal hair and skin growth in mice.

Transit-amplifying cells are crucial for tissue repair and can contribute to cancer when they malfunction.

Certain signals are important for reducing specific chemical markers on hair follicle stem cells during rest periods, which is necessary for healthy hair growth.

Immune cells help regulate hair growth, and better understanding this can improve hair loss treatments.

Smad4 is important for healthy hair follicles because it helps produce a protein needed for hair to stick together and grow.

Endoglin is crucial for proper hair growth cycles and stem cell activation in mice.

DHT stops hair regrowth in mice, similar to human hair loss.