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Mutations in the PTPRQ gene cause significant balance issues in mice due to hair bundle defects in the inner ear.

Protein tyrosine kinases are key in male pattern baldness, affecting skin structure, hair growth, and immune responses.

Protein profiling of forehead skin can help distinguish between frontal fibrosing alopecia and androgenetic alopecia.

Turning white fat into brown-like fat could help fight obesity and type 2 diabetes.

Eating less sugar, milk, and saturated fats and more vegetables and fish may help treat and prevent acne.

Understanding how cells die in the skin is important for treating skin diseases and preventing hair loss.

Blood tests can help understand the genetic differences in people with alopecia areata, including how severe it is and if it's inherited.

Hydrogen sulfide disrupts protein function and root hair growth in plants by modifying proteins.

Ptprq has multiple forms that change during inner ear development.

Human hair follicles produce and respond to erythropoietin, helping protect against stress.

Etifoxine, an anxiety drug, can lessen brain inflammation and cognitive issues in mice, partly by increasing production of protective brain steroids.

The study identified 12 potential biomarkers for hair loss and how they affect hair growth.

Finasteride in male rats causes liver and metabolic issues in their offspring.

Enzymes called PADIs play a key role in hair growth and loss.

Connarus semidecandrus Jack extract promotes hair growth and thickness, reduces prostate cancer cell growth, and could potentially be used as a treatment for hair loss.

A new mouse mutation causes skin and hair defects due to a gene change.

Androgenetic alopecia involves immune cell disruptions, especially increased CD4+ T cells around hair follicles.

GLI1 might protect against the start of skin cancer and is not linked to cancer severity.

Dermal factors are crucial in regulating melanin production in skin.

The conclusion is that androgenetic alopecia and senescent alopecia have unique gene changes, suggesting different causes and potential treatments for these hair loss types.

Chromium supplements don't help with weight loss or improve hormone and metabolism issues in people with polycystic ovary syndrome.

Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

The conclusion is that Treg-targeted therapies have potential, but more knowledge of Treg biology is needed for effective treatments, including for cancer.

Mutant mice help researchers understand hair growth and related genetic factors.

Mice skin has components that could help with hair growth and might be used for diabetes treatment.

Sp6 promotes tooth development by reducing follistatin levels.

The combined treatment with engineered bacteria and yellow LED light improved wound healing in mice.

The document concludes that the development of certain tumors is influenced by genetic background and that a specific gene modification can lead to tumor regression and reduced growth.

Alopecia areata is an autoimmune condition causing hair loss, linked to genetic factors and immune system issues, with no cure yet.

Patients with RASopathies have a higher risk of autoimmune disorders and should be routinely screened.