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Small proline-rich proteins and trichohyalin help make epithelial tissues tougher and more flexible.

Trichohyalin-like proteins are essential for the development of skin structures like hair, nails, and feathers.

A specific chemical change in the S100A3 protein leads to the formation of a four-part structure important for hair formation.

Notch signaling is essential for healthy skin and hair follicle maintenance.

Cat claws stay sharp by shedding their outer layer through microcracks formed during activities.

Understanding genetic mutations helps diagnose and treat skin disorders like ichthyosis.

Notch signaling disruptions can cause various skin diseases.

The harlequin ichthyosis mouse mutation causes thick skin and early death, resembling a human skin disorder.

TGM3 is important for skin and hair structure and may help diagnose cancer.

A new mutation in the ST14 gene broadens the understanding of ichthyosis-hypotrichosis syndrome.

Matriptase is highly active in hair follicles and sebaceous glands, especially during hair growth phases.

Caspases are enzymes important for both cell death and various non-lethal cell functions, affecting head development and hair growth, with different caspases playing specific roles.

Cell-based models help test if cosmetic ingredients really work for hair growth and skin health.

Keratin protein production in cells is controlled by a complex system that changes with cell type, health, and conditions like injury or cancer.

Understanding keratinization is crucial for treating skin conditions like ichthyoses and psoriasis.

Vitamin D receptor is crucial for skin wound healing.

Different types of cell death affect skin health and inflammation, and understanding them could improve treatments for skin diseases.

Steroid sex hormones activate matriptase in prostate cancer cells but not in breast cancer cells.

The skin has multiple layers and cells, serves as a protective barrier, helps regulate temperature, enables sensation, affects appearance, and is involved in vitamin D synthesis.

Recent findings suggest that genetic factors, immune system issues, and skin cell defects might contribute to the development of hidradenitis suppurativa.

Hair growth environment recreated with challenges; stem cells make successful skin organoids.