Search

everythinglearnresearchcommunity

for

Search:↓

Efficient delivery systems are needed for the clinical use of CRISPR-Cas9 gene editing.

Fetal wound healing changes with development, affecting inflammation and collagen, which may influence scarring.

Stem cell niches regulate the number and function of stem cells in the body.

Inflammation helps stem cells repair tissue by directing their behavior.

Fibromodulin might help reduce scarring if increased in adult wounds like in fetal skin that heals without scars.

Hair growth phase and certain genes can speed up wound healing, while an inflammatory mediator can slow down new hair growth after a wound. Understanding these factors can improve tissue regeneration during wound healing.

Msx and Dlx genes are crucial for development, controlling cell behaviors like growth and differentiation through their roles as gene regulators.

Tributyltin exposure causes sex changes, sterilization, and decline in the snail Ocinebrina aciculata, risking its extinction.

Fetal skin heals without scarring due to unique cells and processes not present in adult skin healing.

Artificially inducing hair regrowth in mice can change the normal pattern and timing of hair growth, with minimal color differences between old and new fur.

Hydrogen peroxide can cause scars by changing healing processes and increasing certain protein levels.

Fetal cells could improve skin repair with minimal scarring and are a potential ready-to-use solution for tissue engineering.

Adrenal disorders can cause lasting brain and behavior issues in children.

Mice lacking fibromodulin have disrupted healing patterns, leading to abnormal skin repair and scarring.

Fetal wounds heal without scarring because of different biological factors, which could help improve adult wound healing.

Wounds heal without scarring in early development but later result in scars, and studying Wnt signaling could help control scarring.

Three specific proteins can turn adult skin cells into hair-growing cells, suggesting a new hair loss treatment.

Treatment during development affects hormone balance and sexual behavior in male rats.

Fat stem cells from diabetic mice can still help heal wounds.

Acceptance reduces hair loss distress and medical consultations; coping mechanisms increase them.

Male rats have more somatostatin neurons than females due to testosterone converting to estrogen during early development.

The enzymes 5α-reductase and 3α/β-hydroxysteroid oxidoreductase help create brain-active substances from progesterone and testosterone, which could be used for treatment, but more research is needed to ensure their safety and effectiveness.

Finasteride exposure harms tadpole reproduction and hormone balance.

Keratinocytes show more TGF-β system activity and collagen production as they age, which might affect wound scarring.

Monotreme hair structure and protein distribution are similar to other mammals, but their inner root sheath cornifies differently, suggesting a unique evolution from reptile skin.

Targeting the actin cytoskeleton could improve skin healing and reduce scarring.

Finasteride affects brain stress and enzyme activity differently in various regions, possibly helping with liver-related brain issues.

Hair follicles and deer antlers regenerate similarly through stem cells and are influenced by hormones and growth factors.

Finasteride blocks deoxycorticosterone's anticonvulsant effects in infant rats, but indomethacin doesn't.

Researchers found 617 genes that behave differently in cashmere goat hair follicles, which could help understand hair growth.