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A specific gene variation is linked to a higher risk of polycystic ovary syndrome in Caucasian women.

CAP1 helps Arabidopsis plants grow better under ammonium stress.

The HtrA1L364P mutation causes brain dysfunction and blood vessel damage.

Ptch2 plays a key role in controlling stem cell function and the ability to regenerate after birth.

Wool fiber curliness is linked to the presence of certain proteins and K38.

PRX102 is essential for rice root hair growth by helping transport substances to the tips.

Kir6.1 mutations in Cantú syndrome increase channel sensitivity and hyperpolarization, while SUR2B mutations do not.

Aging reduces stem cell activation, leading to hair loss in mice lacking a specific enzyme.

OsPRX83 helps rice survive stress by improving stress response and antioxidant activity.

Suppressing very long chain fatty acids is linked to skin cancer.

Four botanical extracts were found to quickly attract neutrophils to wounded skin in zebrafish.

Controlling Tslp can improve health in AEC syndrome patients.

Vitamin D affects Msx1 protein expression and may influence mineralized tissue health.

Two mouse mutants have defective hair cuticle cross-linking.

Msx2 deficiency in mice leads to bone growth and organ development problems.

β-catenin in the dermal papilla is crucial for normal hair growth and repair.

Early development of hair, teeth, and glands involves specific signaling pathways and cellular interactions.

ERULUS controls root hair growth by regulating cell wall composition and pectin activity.

New treatments targeting specific genes show promise for treating keratin disorders.

Restoring mitochondrial function in mice reversed their skin wrinkling and hair loss.

Mice lacking a key DNA methylation enzyme in skin cells have a lower chance of activating stem cells necessary for hair growth, leading to progressive hair loss.

Three genes in Arabidopsis are important for plant growth and development by affecting sugar attachment to proteins.

A gene mutation in mice causes skin defects and early death.

Cyclic nucleotide-gated channels are crucial for proper root hair growth and calcium balance in plants.

Mutations in the LSS gene cause a rare type of hereditary hair loss.

Skin tumor regression is helped by retinoic acid signaling blocking Wnt signaling.

Disrupting Smad4 in mouse skin causes early hair follicle stem cell activity that leads to their eventual depletion.

Most Hairless gene mutations reduce its ability to work with the Vitamin D Receptor, which might explain a certain type of hair loss.

Researchers created a new mouse model for studying Citrullinemia Type I and similar conditions, showing symptoms and treatment responses like those in humans.

Hairless protein is crucial for healthy skin and hair, and its malfunction can cause hair loss.