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Targeting mTOR in myeloid cells may help reduce psoriasis symptoms.

Acne patients have higher mTOR gene expression.

Raptor and Rictor have stable expression in hair cycles, with Raptor marking stem cells and Rictor involved in hair shaft formation.

The mTOR signaling pathway might play a role in radiation dermatitis, but more research is needed.

Cepharanthine helps reduce cell aging caused by the cancer drug Doxorubicin by boosting cell cleanup processes.

Removing REDD1 in mice increases skin fat by making fat cells larger and more numerous.

Dihydrotestosterone (DHT) increases oil production in skin cells by activating mTOR, and mTOR inhibitors can reduce this effect.

Certain inhibitors slow down plant growth by causing early cell specialization without changing the cell development pattern.

Certain natural and synthetic compounds may help treat inflammatory skin diseases by targeting a specific signaling pathway.

Exosomes from special stem cells help treat ulcerative colitis by reducing inflammation and stress.

Keeping human skin stem cells is easier with low temperatures and mTOR inhibition.

Lepidium sativum seed extracts helped reduce inflammation and improve insulin response in obese rats on a high-fat diet.

The mTOR pathway may be involved in the development of hair follicle tumors, with higher activity in malignant tumors.

The document concludes that DNA methylation and the mTOR pathway are important for stem cell function and could impact disease treatment.

The April 2016 JEADV issue covered various dermatology topics, including psoriasis, psoriatic arthritis, mTOR inhibitors, autoimmune diseases, photodynamic therapy, viral DNA in skin diseases, chronic hand eczema, and female hair loss.

Cancer treatments targeting specific cells and the immune system can cause skin, mouth, hair, and nail problems, affecting patients' quality of life and treatment adherence.

Copper-doxorubicin nanoparticles effectively treat tumors with less toxicity.

Eating a high-glycemic diet may worsen acne by increasing certain protein levels and expressions in the skin.

Targeted cancer therapies often cause skin problems that need careful management to improve patient quality of life and treatment success.

New therapies for Birt–Hogg–Dubé syndrome are being developed based on understanding the FLCN gene's role.

Studying premature aging syndromes helps understand human aging and suggests potential treatments.

Disrupting the Flcn gene in mice causes early kidney cysts and tumors, which can be treated with rapamycin.

A protein complex called mTORC1 likely affects when hair growth starts in mice.

The document concludes that hair curvature can be explained by the growth patterns caused by the shape and separation of cells in the hair follicle and is affected by specific molecular pathways.

Dihydrotestosterone (DHT) doesn't affect rat skin cell growth, but it does change cell cycle, protein levels, and other cell functions, potentially shortening hair growth cycle.

Certain cells outside the hair follicle's bulge area can quickly regenerate damaged hair follicles, potentially helping to reduce hair loss from cancer treatments.

The study found that analyzing certain cell signaling pathways is not a reliable method to tell apart two types of skin tumors.

Skin problems from transplant drugs are common and need careful management in organ transplant patients.

Birt–Hogg–Dubé syndrome is a rare genetic condition causing skin lesions, lung cysts, and a higher chance of kidney cancer.

Eating fewer calories may slow skin aging and improve skin health through various biological changes.