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The Rotterdam Study found risk factors for elderly diseases, links between lifestyle and genetics with health conditions, and aimed to explore new areas like DNA methylation and sensory input effects on brain function.

The circadian clock affects skin stem cell behavior, impacting aging and cancer risk.

Some drugs can cause rare but potentially deadly skin reactions, and early treatment and avoiding the drug again are key.

The Rotterdam Study aims to understand disease causes in the elderly and has found new risk factors and genetic influences on various conditions.

Teriflunomide helps reduce multiple sclerosis symptoms and is safe for most patients.

Wnt1/βcatenin signaling is crucial for heart repair after injury.

Hair loss and aging are caused by the breakdown of a key protein in hair stem cells.

Fat cells help recruit healing cells and build skin structure during wound healing.

Adding anti-Müllerian hormone to PCOS criteria lowers the number of women diagnosed.

Biomaterials with MSC-derived substances could improve tissue repair and have advantages over direct cell therapy.

Skin needs dermal β-catenin activity for hair growth and skin cell multiplication.

Regenerated fully functional hair follicles using stem cells, with potential for hair regrowth therapy.

Different types of skin cells have unique genetic markers that affect how likely they are to spread cancer.

Some skin medications are safe during pregnancy and breastfeeding, but others can harm the baby and should be avoided.

Stress-related substance P may lead to hair loss and negatively affect hair growth.

Early diagnosis and individualized treatment improve outcomes for Congenital Adrenal Hyperplasia.

The document concludes that accurate diagnosis of ichthyosis is crucial for treatment and genetic advice, and ongoing research is needed for better therapies.

The Sonic hedgehog pathway is crucial for new hair growth during mouse skin healing.

Aging causes skin stem cells to work less effectively.

Sonic hedgehog signaling is crucial for hair growth and maintaining hair follicle identity.

Prolactin contributes to hair loss by promoting hair follicle shrinkage and cell death.

Fat cells in the skin help start healing and form important repair cells after injury.

The research identified various cell types in mouse and human teeth, which could help in developing dental regenerative treatments.

Stress increases nerve fibers and immune cell activity in mouse skin, possibly worsening skin conditions.

Stress increases a factor in mice that leads to hair loss, and blocking this factor may prevent it.

Lampreys have a functional vitamin D receptor that may help detoxify harmful substances.

Cystatin M/E strongly inhibits cathepsin V and cathepsin L, important for skin formation.

Androgens block hair growth by disrupting cell signals; targeting GSK-3 may help treat hair loss.

The Wnt/β-catenin pathway helps tissue regeneration but can also cause fibrosis, and drugs that inhibit this pathway may aid in healing skin and heart tissues.

Oxidative stress affects hair loss in men with androgenetic alopecia.