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Key genes linked to immune response are upregulated in hair follicles and skin tissues in chronic discoid lupus erythematosus.

Researchers developed a new way to measure gene activity in single hair follicles and found that a specific gene's activity changes with different amounts and times of treatment.

Male-pattern hair loss is largely influenced by genetics, with key genes identified.

Topical Imiquimod may fight vascular tumors by affecting blood vessels or the immune system, low iron might be linked to some hair loss, removing the top skin layer helps vitamin C get in, genetic testing helps diagnose skin conditions, and too much iron could worsen skin inflammation.

Specific immune cells cause alopecia areata and blocking certain proteins can prevent it.

Enz_MoriL from mulberry leaves helps hair growth by affecting specific cell pathways.

Trichloroethylene causes skin inflammation in mice by increasing certain immune proteins.

TAGX-0003 protected hair follicles and reversed alopecia areata in a mouse model.

JAK inhibitors like tofacitinib may effectively treat Alopecia Areata.

Vitamin D helps protect skin, PSORS1 gene's risk interval for psoriasis is expanded, hair follicles can be generated from mouse cells, and interferon-γ may cause pigmented skin lesions.

Crataegi Fructus water extract helps hair grow better and thicker.

Blocking TYK2 might be a new way to treat hair loss from alopecia areata.

Apremilast did not work for treating severe alopecia areata.

Cytotoxic T cells cause hair loss in chronic alopecia areata.

The combination therapy of PEG-IFN α-2a and Ribavirin can cause widespread hair loss and skin reactions.

Hair follicle stem cells reduced hair loss and inflammation in mice with a condition similar to human alopecia.

IL-17 and certain immune cells are linked to more severe alopecia areata.

Mycophenolate helps reverse hair loss effects caused by IFN-γ by activating a key hair growth pathway.

IL-24 contributes to skin aging by increasing collagen-degrading enzymes and decreasing collagen production when exposed to air pollution.

Targeting specific T cells may help treat alopecia areata.

Alopecia areata is caused by immune system issues, and JAK inhibitors might help treat it.

Frontal fibrosing alopecia shows increased inflammation and JAK-STAT pathway activity without reduced hair proteins.

A patient with Alopecia Areata had complete hair regrowth after using the drug baricitinib.

Sorafenib may cause hair loss in a way similar to alopecia areata.

Oral tofacitinib shows promise in treating atopic dermatitis and alopecia areata, but only slight improvement in vitiligo.

The humanized AA mouse model is better for testing new alopecia areata treatments.

ILC1-like cells can independently cause alopecia areata by affecting hair follicles.

ILC1-like cells may contribute to hair loss in alopecia areata and could be new treatment targets.

A patient with alopecia areata regrew hair after taking tofacitinib and showed changes in certain blood and skin markers.

Both vitiligo and alopecia areata involve an immune response triggered by stress and specific genes, with treatments targeting this pathway showing potential.