Search

everythinglearnresearchcommunity

for

Search:↓

Skin problems in COVID-19 patients are rare and may be due to the body's complex immune response or blood clotting issues.

Both vitiligo and alopecia areata involve an immune response triggered by stress and specific genes, with treatments targeting this pathway showing potential.

Stress may trigger hair loss by affecting immune protection in hair follicles.

People with late-stage HIV-1 often experience a specific type of hair loss linked to multiple factors, including nutritional issues and immune responses.

Accurate diagnosis is key for treating different kinds of hair loss, and immune response variations may affect the condition and treatment results.

People with atopic dermatitis are more likely to develop other skin conditions due to shared genetics and immune pathways.

CRAC channels are crucial for the development and function of specialized immune cells, preventing severe inflammation and autoimmune diseases.

Alopecia areata severity and treatment response are linked to specific cytokine levels.

Hair loss involves immune responses, inflammation, and disrupted signaling pathways.

CRTH2 antagonists might be useful for treating many conditions because they play a role in immune and inflammation responses.

Human body's immune cells are more common in the layer of fat just beneath the skin than in deeper fat layers.

The cause of alopecia areata is likely a mix of genetics, immune system issues, and environmental factors, with more research needed to understand it fully.

Hair follicles are essential for skin health, aiding in hair growth, wound healing, and immune function.

Men and women get COVID-19 at similar rates, but men tend to get sicker and have a higher risk of dying, while women usually have stronger immune responses and vaccine reactions.

The document concludes that immune system abnormalities cause alopecia areata, but the exact process is still not completely understood.

A boy's growth and immune problems were caused by a new mutation in the STAT5B gene.

Targeting immune tolerance issues in Alopecia Areata could restore hair growth and maintain remission.

Four specific genes are linked to keloid formation and could be potential treatment targets.

Skin abnormalities can indicate immunodeficiency due to shared origins with the immune system.

Rhododendrol in skin-whitening products can cause skin depigmentation and immune reactions.

The document concludes that the immune-inhibitory environment of the hair follicle may prevent melanoma development.

UV light helped human hair transplants survive in mice without broad immunosuppression.

Hair graying is influenced by factors like nerves, fat cells, and immune cells, not just hair follicles.

Pine bark extract helps mice grow hair by reducing inflammation and boosting growth factors.

Ingenol mebutate gel changes gene expression related to skin development and immune response in actinic keratosis.

Certain genetic variants linked to immune response increase the risk of alopecia areata in Taiwanese people.

Oxidative stress and immune dysfunction are linked to both Hashimoto's thyroiditis and polycystic ovary syndrome, with diet and specific treatments important for managing these conditions.

A new mutation in the TMEM173 gene and a risk allele in IFIH1 cause a unique set of immune-related symptoms.

Androgenetic alopecia involves immune cell disruptions, especially increased CD4+ T cells around hair follicles.

Hemp seed biomaterials may reduce hair loss and improve hair growth.