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Keloid skin disorder involves abnormal fibroblast activation and immune response, linked to a group of genes including FGF11.

A specific enzyme is essential for proper hair follicle stem cell development and healthy skin.

Jagged-1 in skin Tregs is crucial for timely wound healing by recruiting specific immune cells.

Inflammation plays a key role in activating skin stem cells for hair growth and wound healing, but more research is needed to understand how it directs cell behavior.

Four specific genes are linked to keloid formation and could be potential treatment targets.

Different cell types work together to repair skin, and targeting them may improve healing and reduce scarring.

Understanding the immune-related causes of Alopecia Areata has led to potential treatments like JAK inhibitors.

The document concludes that the skin's immune system is complex, involving interactions with hair follicles, nerves, and microbes, and can protect or cause disease, offering targets for new treatments.

Special immune cells called Treg cells are important for maintaining and regenerating hair by activating a specific growth signal in hair stem cells.

Different stem cells are key for hair growth and health, and understanding their regulation could help treat hair loss.

Acne patients' skin, both with and without lesions, shows a strong immune response and higher antimicrobial activity.

Stress may trigger hair loss by affecting immune protection in hair follicles.

KAP-depleted hair causes less immune response and is more biocompatible for implants.

TGFβ's manipulation of inflammation and immune cells affects cancer spread, suggesting new treatment strategies and biomarkers.

Activating Notch in adult skin causes T cells and neural crest cells to gather, leading to skin issues.

Alopecia areata involves persistent gene abnormalities and immune activity, even in regrown hair, suggesting a risk of relapse.

The skin's internal clock affects healing, cancer risk, aging, immunity, and hair growth, and disruptions can harm skin health.

Prolactin affects hair growth and skin conditions, and could be a target for new skin disease treatments.

Blocking JAK/STAT pathways can help treat hair loss from alopecia areata.

Vitamin A may influence hair loss conditions like alopecia, but more research is needed to understand how.

The cause of Frontal fibrosing alopecia, a type of hair loss, is complex, likely involving immune responses and genetics, but is not fully understood.

Dermal macrophages might help regrow hair.

miR-486 may help prevent hair loss in alopecia areata.

Certain immune cells contribute to skin autoimmune diseases, and some treatments can reverse hair loss in these conditions.

Certain microRNAs are linked to various skin diseases and could be used to diagnose and treat these conditions.

Deleting Snai2 and Snai3 causes fatal autoimmunity.

Lymphocytes, especially CD8+ T cells, play a key role in causing alopecia areata, and targeting them may lead to new treatments.

Lysophospholipids like LPA and S1P are important for hair growth, immune responses, and vascular development, and could be targeted for treating diseases.

Sebaceous glands play a key role in skin health, immunity, and various skin diseases.

ILC1-like cells can cause alopecia areata by attacking hair follicles.