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Cells from the lower part of hair follicles are a promising, less invasive option for immune system therapies.

Monocytes might be linked to hair loss after COVID-19.

CD117 and platelet-derived growth factor receptor α may play a role in alopecia areata.

IFN-γ and IL-2 are important for T cell activation in hair loss in mice.

Targeting specific T cells may help treat alopecia areata.

The new aptamer TAGX-0003 shows promise as an effective treatment for hair loss disorder alopecia areata.

TAGX-0003 protected hair follicles and reversed alopecia areata in a mouse model.

IL-17 and certain immune cells are linked to more severe alopecia areata.

Hair follicles are normally protected from the immune system, but when this protection fails, it can cause hair loss in alopecia areata.

Apremilast did not work for treating severe alopecia areata.

T-cells in alopecia areata scalp show abnormal regulation, leading to less inflammation.

As skin cells mature, vitamin D receptor levels decrease while retinoid X receptor α levels increase.

Antroquinonol may help prevent skin depigmentation by suppressing certain immune cells.

Alopecia areata is caused by immune system issues, and JAK inhibitors might help treat it.

Ginsenoside Rg1 protects mouse skin from UVB damage and helps control inflammation.

Sorafenib may cause hair loss in a way similar to alopecia areata.

Chronic inflammatory skin diseases are caused by disrupted interactions between skin cells and immune cells.

Frontal fibrosing alopecia shows increased inflammation and JAK-STAT pathway activity without reduced hair proteins.

Fetal skin has unique immune cells different from adult skin.

Higher levels of certain DNAs in blood may indicate hair follicle damage in alopecia areata patients.

Blocking the protein CXCL12 with a specific antibody can increase hair growth in common hair loss conditions.

Some multiple sclerosis treatments may trigger hair loss conditions like alopecia areata.

Booster shots of mRNA vaccines for COVID-19 increased protective antibodies without worsening autoimmune skin conditions in patients.

JAK inhibitors help hair growth by boosting beta-catenin activity in hair root cells.

Type 1/17 inflammation in psoriasis increases skin cell growth due to a molecule that could be a new treatment target.

ILC1-like cells may contribute to hair loss in alopecia areata.

ILC1-like cells can independently cause alopecia areata by affecting hair follicles.

ILC1-like cells may contribute to hair loss in alopecia areata and could be new treatment targets.

Tofacitinib regrew hair in a man with total hair loss but raised cytokine levels, needing more research on possible side effects.

Innate lymphoid cells type 1 may contribute to alopecia areata.