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Despite progress in treatment, the exact cause of Alopecia areata is still unknown.

Certain HLA class II alleles increase or decrease the risk of alopecia areata.

Hair loss in Rhesus macaques may be caused by a skin allergy-related condition.

The 2015 Hair Research Congress concluded that stem cells, maraviroc, and simvastatin could potentially treat Alopecia Areata, topical minoxidil, finasteride, and steroids could treat Frontal Fibrosing Alopecia, and PTGDR2 antagonists could also treat alopecia. They also found that low-level light therapy could help with hair loss, a robotic device could assist in hair extraction, and nutrition could aid hair growth. They suggested that Alopecia Areata is an inflammatory disorder, not a single disease, indicating a need for personalized treatments.

Dandruff is linked to increased T cells and weakened immune protection in hair follicles.

Lymphocytes, especially CD8+ T cells, play a key role in causing alopecia areata, and targeting them may lead to new treatments.

A protein called EGFR protects hair follicle stem cells, and when it's disrupted, hair follicles can be damaged, but blocking certain pathways can restore hair growth.

Men generally have more severe COVID-19 cases and higher death rates than women due to biological differences.

The review suggests that other immune cells besides CD8+ T cells may contribute to alopecia areata and that targeting regulatory cell defects could improve treatment.

Early stage bald spots are linked to skin inflammation and damage to the upper part of the hair follicle.

Alopecia areata is an autoimmune condition causing hair loss, influenced by genetics, stress, and diet, and may be prevented by a high soy oil diet.

Alopecia areata can be reversed by JAK inhibitors, promoting hair regrowth.

Alopecia areata is caused by immune system attacks on hair follicles, often triggered by viral infections.

Mice treatments didn't grow hair, a patient treatment may affect immune response, and people with hair loss often feel anxious or depressed.

The cause of alopecia areata is likely a mix of genetics, immune system issues, and environmental factors, with more research needed to understand it fully.

Alopecia Areata has no guaranteed treatment for hair regrowth, but options like corticosteroids and minoxidil are used, with future research focusing on genetic and immune therapies.

Transplant success has improved with better immunosuppressive drugs and donor matching.

Using low-dose IL-2 to increase regulatory T cells might be a safe way to treat type 1 diabetes without severe side effects.

The document concludes that Cutaneous Lupus Erythematosus has different forms, is influenced by genetic and environmental factors, and can be treated with various medications, but more targeted therapies are needed.

Somatostatin may help protect hair follicles from immune attacks.

The document concludes that better targeted treatments are needed for wound healing, and single-cell technologies may improve cell-based therapies.

New treatments for immune disorders caused by FOXN1 deficiency are promising.

Newborn screening and gene therapy are expected to improve outcomes for Omenn syndrome patients.

Thymus transplantation successfully restored immune function in infants with FOXN1 deficiency.

MILL molecules are unique immune proteins in mice that don't need TAP to appear on cell surfaces.

Oral lichen planus is a chronic disease causing mouth discomfort and sometimes needs immunosuppressive treatment.

Plasmacytoid dendritic cells are a key factor in causing hair loss in alopecia areata and could help differentiate it from other hair loss conditions.

A genetic marker linked to a type of hair loss was found in most patients studied.

Understanding how hair follicle stem cells work can help find new ways to prevent hair loss and promote hair growth.

Tofacitinib temporarily regrew hair in a man with alopecia, but its effects didn't last.