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Certain genetic variants are linked to frontal fibrosing alopecia in Spanish patients.

Certain genes are linked to the risk of developing Alopecia Areata.

Mercury allergy linked to specific genes may contribute to burning mouth syndrome, and silicon might play a role in maintaining healthy hair.

Certain HLA class II alleles increase or decrease the risk of alopecia areata.

HLA-DRB5 and other genes may be linked to alopecia universalis.

Alopecia areata involves both innate and adaptive immunity, with specific genes linked to the disease.

Half of people with Alopecia Areata may see hair regrowth within a year without treatment, but recovery is unpredictable.

Alopecia areata, a common autoimmune hair loss condition, often runs in families.

Alopecia areata is an autoimmune disease where T cells attack hair follicles.

Alopecia areata is an autoimmune condition causing hair loss due to the immune system attacking hair follicles, often influenced by genetics and stress.

Alopecia areata is an autoimmune disease causing hair loss, treatable with immune-modulating drugs, and linked to genetics.

Men generally have more severe COVID-19 cases and higher death rates than women due to biological differences.

Alopecia areata is likely caused by a combination of genetic factors and immune system dysfunction, and may represent different diseases with various causes.

Researchers found a specific immune receptor in patients that causes severe skin reactions to a drug.

The conclusion is that individual differences in COVID-19 severity are influenced by factors like age, sex, race, and genetics, which are important for personalized medicine.

Alopecia Areata has no guaranteed treatment for hair regrowth, but options like corticosteroids and minoxidil are used, with future research focusing on genetic and immune therapies.

Despite progress in treatment, the exact cause of Alopecia areata is still unknown.

More research is needed to understand the genetic causes of Alopecia areata to develop better treatments.

Some patients with mycosis fungoides or Sézary syndrome experience hair loss, which may be similar to alopecia areata or linked to skin lesions, possibly due to abnormal T cells, and bexarotene can help treat it.

The DQB1*03 allele is linked to higher alopecia areata risk in Italians.

FOXN1 gene mutations cause a rare, severe immune disease treatable with cell or tissue transplants.

Alopecia areata is linked to thyroid autoimmunity but not type 1 diabetes.

Thymic transplantation normalized some T-cells but not others, maintaining immune function.

Patients with Alopecia areata have fewer specific immune cells that normally regulate the immune system, which may contribute to the condition.

The document concludes that ongoing research using animal models is crucial for better understanding and treating Alopecia Areata.

Genetic factors alone might not cause pemphigus vulgaris; other factors like birth complications and puberty may trigger it.

The cause of alopecia areata is likely a mix of genetics, immune system issues, and environmental factors, with more research needed to understand it fully.

The document concludes that pregnancy and cancer share immune evasion tactics, but more research is needed before using checkpoint blockade immunotherapy in pregnant cancer patients to avoid harm to the placenta.

Alopecia areata has a complex genetic basis that was not fully understood as of 2001.