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The p.P25L mutation in the KRT5 gene causes a rare skin condition that worsens over time and may lead to hair loss starting in young adulthood.

Mutations in three genes cause Uncombable Hair Syndrome, leading to frizzy hair that can't be combed flat.

Mutations in TBX3 cause horses to have more even hair color instead of Dun camouflage.

The transition from growth to regression in Cashmere goat hair follicles involves changes in expression of genes related to keratin and cell differentiation.

ODC overexpression in hair cells increases tumor growth by reducing Notch signaling.

A device was made in 2008 to measure hair loss severity. Other findings include: frizzy mutation in mice isn't related to Fgfr2, C/EBPx marks preadipocytes, Cyclosporin A speeds up hair growth in mice, blocking plasmin and metalloproteinases hinders healing, hyperbaric oxygen helps ischemic wound healing, amniotic membranes heal wounds better than polyurethane foam, rhVEGF165 from a fibrin matrix improves tissue flap viability and induces VEGF-R2 expression, and bFGF enhances wound healing and reduces scarring in rabbits.

Sphynx cats are hairless and Devon Rex cats have curly hair due to specific genetic mutations.

A woman with a new PTCH gene mutation has both Gorlin syndrome and severe hair loss.

Researchers made a mouse model with curly hair and hair loss by editing a gene.

A Taiwanese patient had hair loss and skin bumps without the usual gene mutation, suggesting other genetic factors might be involved.

Keratosis pilaris is a common skin condition where hair follicles get clogged with keratin, mostly on the arms and thighs.

Porokeratotic eccrine and hair follicle nevus is a very rare skin disorder possibly caused by a GJB2 gene mutation.

Researchers found a new mutation in the HR gene linked to a rare hair loss condition.

A KRT71 mutation in Hereford cattle in Uruguay causes thin, curly hair and scaly skin.

Uncombable hair syndrome causes frizzy hair and can affect the nervous system, eyes, and ears, often co-occurring with other hair, skin, nail, and teeth conditions, and is linked to three specific gene mutations.

Mutations in the Vitamin D receptor gene can cause hair loss similar to mutations in the Hairless gene.

A Gsdma3 mutation causes hair loss due to stem cell damage from skin inflammation.

Keratosis pilaris is often linked to genetic mutations and causes skin and hair abnormalities, regardless of those mutations.

A TP63 gene mutation causes significant hair loss and mild skin, nail, and tooth abnormalities.

The 2004 hair research meeting presented new findings on hair cell differentiation, genetic factors in hair loss, hair pigmentation, and potential targeted therapies.

The document concludes that mouse models are crucial for studying hair biology and that all mutant mice may have hair growth abnormalities that require detailed analysis to identify.

Blocking a specific protein signal can make hair grow on mouse nipples.

Monotreme hair structure and protein distribution are similar to other mammals, but their inner root sheath cornifies differently, suggesting a unique evolution from reptile skin.

A specific type of skin cell creates an opening for hair to grow out, and problems with this process can lead to skin conditions.

The Megsin-Cre transgene is a new tool for genetic manipulation in the skin and upper digestive tract.

The document concludes that hair loss is influenced by genetics and other factors, and while treatments like finasteride can help, they have limitations and side effects.

VEGFR-2 activation is likely involved in hair follicle growth, survival, and development.

A mother and daughter have a rare genetic hair loss disorder with no effective treatment.

FGF-5 promotes Cashmere goat hair growth by increasing keratin genes and reducing certain LncRNA and target genes.

The document concludes that Borrelia afzelii causes a skin condition in France, a gene is linked to hair loss in Caucasian women, and various genetic mutations affect skin diseases.