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The document concludes that immune system abnormalities cause alopecia areata, but the exact process is still not completely understood.

High mTORC1 activity slows hair growth and causes it to lose color.

The document concludes that accurate diagnosis and appropriate management are crucial for treating various hair disorders, which have significant psychological impacts.

The research found that a specific skin cell type not only triggers hair growth but also controls hair color, and that aging can lead to hair loss and color changes.

The study concluded that genetic mutations affect human hair diseases and identified key genes and pathways involved in hair growth and cycling.

New treatments targeting insulin, blood flow, and inflammation could improve hormone-related hair conditions with fewer side effects.

Androgens can both promote and prevent hair growth due to differences in gene expression in hair follicles.

Different hair growth problems are caused by genetic issues or changes in hair growth cycles, and new treatments are being developed.

New protein changes may be involved in the immune attack on hair follicles in alopecia areata.

Cadmium chloride pollution can cause skin disorders, speed up aging, and prevent hair growth.

Recent discoveries have improved our understanding of hair loss, but challenges in treatment and knowledge among specialists still exist.

Schizophrenia patients' stem cells show abnormal neuron development and mitochondrial issues.

The term "porokeratotic adnexal ostial nevus" is proposed to unify overlapping skin conditions involving eccrine and hair follicles.

A mutation in the KRT71 gene causes a hair disorder by disrupting hair follicle structure and texture.

Researchers successfully isolated and identified key stem cells in human hair follicles, which could help develop new skin and hair treatments.

Hair follicle biology advancements may lead to better hair growth disorder treatments.

The document concludes that understanding hair follicle cell cycles is crucial for hair growth and alopecia research, and recommends specific techniques and future research directions.

Sox2 controls hair color by affecting pigment production in hair follicles.

A protein called sFRP4 from skin cells stops the development of pigment-producing cells in hair.

Reducing Tyrosinase prevents mature color pigment cells from forming in mouse hair.

Canine hair follicles have stem cells similar to human hair follicles, useful for studying hair disorders.

Genetically modified stem cells from human hair follicles can lower blood sugar and increase survival in diabetic mice.

The boy with woolly hair nevus had thinner hair and abnormal hair follicles, which improved with treatment but worsened when treatment stopped.

Vitamin D and its receptor are important for hair and scalp health and may help treat hair loss.

Hair patterns in mice are controlled by both a global system dependent on Fz6 and a local self-organizing system.

Histone H4, released by cells exposed to colchicine, can cause hair loss by inhibiting cell growth and enzyme activity.

Hair color is determined by melanin produced and transferred in hair follicles.

Epithelial-mesenchymal interactions control hair growth cycles through specific molecular signals.

The conclusion is that proper signaling is crucial for hair growth and development, and errors can lead to cancer or hair loss.

Wnt ligands are crucial for hair growth and repair.