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FGF-2&D/P nanoparticles can help treat hair loss.

Sesn2 protects inner ear hair cells from damage by regulating certain cell survival pathways.

New hair loss treatments may include topical medications, injections, and improved transplant methods.

Some natural compounds can protect fish ear cells from damage by certain antibiotics without affecting the antibiotics' ability to fight infections.

Obesity and bariatric surgery can cause hair thinning and temporary hair loss due to nutritional deficiencies and stress.

Caffeine can damage hearing cells and affect hearing recovery after ear trauma.

Gene therapy, especially using atoh1, shows promise for creating functional sensory hair cells in the inner ear, but dosing and side effects need to be managed for clinical application.

Humans have limited regenerative abilities, but new evidence shows the adult brain and heart can regenerate, and future treatments may improve this by mimicking stem cell environments.

EGFR helps control how hair grows and forms without needing p53 protein.

Mongolian gerbils heal wounds differently than mice, with unique protein levels and gene expression that affect skin repair.

Blocking adenosine A2B receptor may prevent or treat hearing loss.

Transplanted transected hair follicles can survive but grow at unsatisfactory rates and are thinner, suggesting limited potential for unlimited donor hair supply.

Cutting hair follicles into pieces for transplantation results in poor growth and thinner hair, and the technique is more invasive than previously thought.

FGF-1 causes spiral ganglion neurites to branch more.

The new method makes it easier to study the whole cochlea from newborn mice and rats in the lab.

Removing Dicer from pigment cells in newborn mice causes early hair graying and changes in cell migration molecules.

Proteins like aPKC and PDGF-AA, substances like adenosine and ATP, and adipose-derived stem cells all play important roles in hair growth and health, and could potentially be used to treat hair loss and skin conditions.

Aging mice have slower hair regeneration due to changes in signal balance, but the environment, not stem cell loss, controls this, suggesting treatments could focus on environmental factors.

Low-dose radiation affects hair stem cell function and survival by changing their genetic material's structure.

Tet1/2/3 enzymes affect hair follicle cell development by influencing BMP signaling.

The document concludes that for hair and feather growth, it's better to target the environment around stem cells than the cells themselves.

The conclusion suggests that focusing on certain cellular pathways may improve the prevention and repair of hair loss caused by radiotherapy.

Different types of stem cells and their environments are key to skin repair and maintenance.

Skin cells show flexibility in healing wounds and forming tumors, with potential for treating hair disorders and chronic ulcers.

Skin health and repair depend on the signals between skin stem cells and their surrounding cells.

Follistatin and LIN28B together improve the ability of inner ear cells in mice to regenerate into hearing cells.

Stem cells and their surrounding environment in hair follicles work closely together, affecting hair growth and having implications for cancer and tissue regeneration.

The environment around melanocyte stem cells is key for hair regeneration and color, with certain injuries affecting hair color and potential treatments for pigmentation disorders.

Skin's epithelial stem cells are crucial for repair and maintenance, and understanding them could improve treatments for skin problems.