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Certain genetic variants in the androgen receptor are linked to higher PSA levels, potentially affecting prostate cancer screening outcomes.

Rare ULBP3 gene changes may raise the risk of Alopecia areata, a certain FAS gene deletion could cause a dysfunctional protein in an immune disorder, and having one copy of a specific genetic deletion is okay, but two copies cause sickle cell disease.

Organ transplant recipients have a higher risk of skin cancer over time, atopic dermatitis skin shows unusual bacterial and fungal patterns, a new tool for measuring hidradenitis suppurativa severity was created, and gene expression changes in male baldness suggest new treatments.

Correct testosterone levels for age to treat prostate cancer, using low dose treatments as necessary.

Androgen use may increase the risk of stroke, but more research is needed.

Scalp basal cell carcinoma may be more aggressive and harder to treat than other types, requiring special attention and further research.

Alopecia areata is an autoimmune condition causing hair loss, influenced by genetics, stress, and diet, and may be prevented by a high soy oil diet.

The Rotterdam Study updated its design and objectives in 2012, providing insights into various diseases in the elderly, including skin cancer, bone health, liver disease, neurological and psychiatric conditions, and respiratory issues.

New genes found linked to balding, may help develop future treatments.

A gene variant increases the risk of a type of hair loss by affecting hair protein production.

Managing metabolic syndrome needs both lifestyle changes and medical treatments.

Postpartum depression is linked to changes in brain chemicals, inflammation, stress, and certain genes, and can potentially be identified by markers like specific steroids, serotonin levels, and vitamin D levels.

The document concludes that ongoing research using animal models is crucial for better understanding and treating Alopecia Areata.

Longer GGN repeats in the androgen receptor gene are linked to polycystic ovary syndrome.

The document concludes that various findings in rheumatology offer insights into disease severity, treatment responses, and potential risks in medication, with some limitations due to unspecified participant numbers.

Testosterone may influence COVID-19 severity and outcomes.

Men with a family history of hair loss on their mother's side are more likely to have female pattern hair loss.

Male pattern baldness is mostly inherited, involves many genes, and is linked to other traits like early puberty and strong bones.

Women with PCOS face higher risks of diabetes and heart disease, and these risks increase with obesity.

Type 2 diabetes, bacterial scalp infections, and tight hairstyles like braids and weaves are linked to a higher risk of a scarring hair loss condition in African American women.

Genetic variations affect dutasteride treatment response for male pattern hair loss.

A gene variant causes patched hair loss in mice, similar to alopecia areata in humans.

Male pattern baldness is linked to a higher risk of certain skin cancers, especially on the scalp.

Female donor to male recipient sex mismatch and positive ACA-IgG are key risk factors for vitiligo and alopecia areata in chronic GvHD patients.

Young women in West Bengal, India, with PCOS often have estrogen resistance, leptin receptor issues, folate deficiency, T2DM, and acanthosis, commonly linked to obesity.

Tumor cell adhesion is linked to higher risk of SLN metastasis and melanoma recurrence, and a model including these factors predicts these outcomes better than one with just clinical data.

Certain variations of the HDAC9 gene can increase or decrease stroke risk in the Chinese population.

Taking 5-alpha reductase inhibitors does not increase the risk of breast cancer in men.

Tacrolimus causes fewer acute rejections than cyclosporin A in kidney transplants but doesn't necessarily improve kidney function after one year; cardiovascular risks and side effects vary between the two drugs.

The research found genetic differences in identical twins that could explain why one twin has a disease while the other does not.