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CRISPR/Cas9 gene-editing shows promise for improving sheep and goat breeding but faces challenges with efficiency and accuracy.

Cancer development is like natural selection, involving mutated cells and environmental factors.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

The oncoprotein causes abnormal hair growth without increasing skin cancer risk.

The lentiviral array can monitor and predict gene activity during stem cell differentiation.

A gene mutation in mice causes permanent hair loss and skin issues.

FoxN1 gene is essential for proper thymus structure and preventing hair loss.

Male hormones control a specific gene in hamster skin, with different hormones having varying effects.

Higher androgen levels and site-specific AR expression cause sex-related skeletal differences, and certain steroids can boost AR expression and androgen effects in bone cells.

Metallothionein likely helps in cell growth and development in wool follicles of fetal sheep.

Obesity can weaken the skin's ability to fight infections because fat cells stop and reduce the infection-fighting properties of nearby stem cells.

New mutations in the SLC39A4 gene found in twins help understand the genetic cause of acrodermatitis enteropathica.

Epimorphin, a protein, plays a key role in the development of hair follicles in human fetuses, but it doesn't help in maintaining the stem cell population of the follicular skin layer.

Androgen receptors are active in many tissues of both male and female mice, not just reproductive organs.

New treatments targeting skin stem cells show promise for skin repair, anti-aging, and cancer therapy.

Somatostatin may help protect hair follicles from immune attacks.

A new mutation in the Connexin 26 gene was found in a patient with KID syndrome, expanding the known disorders linked to this gene.

Damaged hair follicles make mice more prone to skin inflammation and skin cancer after UV exposure.

The conclusion is that skin and hair patterns are formed by a mix of cell activities, molecular signals, and environmental factors.

Higher IGF-1 levels in hair follicles link to better finasteride results for hair loss.

Fibroblast growth factor receptor signaling controls cell development and repair, and its malfunction can cause disorders and cancer, but it also offers potential for targeted therapies.

Alopecia areata is likely an autoimmune disease with unclear triggers, involving various immune cells and molecules, and currently has no cure.

The document concludes that mouse models are crucial for studying hair biology and that all mutant mice may have hair growth abnormalities that require detailed analysis to identify.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

The hair keratin gene KRT81 is found in both normal and breast cancer cells and helps them invade surrounding tissues.

A WNT10A gene mutation leads to ectodermal dysplasia by disrupting cell growth and differentiation.

A gene network led by RSL4 is crucial for early root hair growth in response to cold in Arabidopsis thaliana.

The gene p53 is crucial for removing damaged cells to allow for healthy tissue renewal.

The CUBIC protocol allows detailed 3D visualization of proteins in mouse skin biopsies.

Oral cyclosporin doesn't stop hair loss.