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The document concludes that patient outcomes for Congenital Adrenal Hyperplasia are often not ideal because of poor management and a need for better diagnosis and treatment methods.

MendelVar is a tool that helps identify important genes by combining GWAS data with Mendelian disease information.

Some skin tumors may start from hair follicle stem cells.

FOXN1 is crucial for thymus development and immune response in Xenopus laevis.

Not having the gene PLAAT3 leads to fat loss, high insulin resistance, and abnormal fat levels in the blood due to a disruption in fat cell development and function.

GTL1 is needed to control root hair growth and prevent problems when there are too many nutrients.

The Arabidopsis rhd2 mutant is more sensitive to drought because of changes in cell membrane proteins and cell structure.

Wnt signaling is crucial for skin development and hair growth.

Progeroid mouse models show signs of early aging similar to humans, helping us understand aging better.

Androgen self-administration might be controlled by membrane receptors, not nuclear ones.

Early diagnosis and treatment of TPP can prevent complications.

Rapamycin may help treat Leigh syndrome by targeting protein kinase C.

Adipocytes can change into fibroblast-like cells to help with wound healing.

Adult esophageal cells can start to become like skin cells, with a key pathway influencing this change.

Sebaceous glands can heal and regenerate after injury using their own stem cells and help from hair follicle cells.

miR-29 is a key factor that accelerates aging.

A protein called FERONIA helps control root hair growth in response to cold and low nitrogen by activating nutrient-sensing pathways in a plant called Arabidopsis.

Hairless mammals have genetic changes in both their protein-coding and regulatory sequences related to hair.

The SYP123-VAMP727 complex is important for transporting materials that harden the root hair shank in Arabidopsis.

The Asiatic lion has very low genetic diversity and unique genetic traits, highlighting the need for its conservation.

Certain cells in the adult mouse ear come from cranial neural crest cells, but muscle and hair cells do not.

NAC1 controls certain enzymes that reduce root hair growth in Arabidopsis.

Activating β-catenin in mammary cells leads to changes that cause early-stage abnormal growths similar to skin structures.

Removing centrosomes from skin cells leads to thinner skin and stops hair growth, but does not greatly affect skin cell differentiation.

Removing Sprouty genes in mice causes various hormone-related issues but does not increase cancer risk by one year of age.

The gene Ascl4 is not necessary for the development of hair, teeth, or mammary glands.

Disrupted cholesterol production impairs hair follicle stem cells, leading to hair loss.

A protein called EGFR protects hair follicle stem cells, and when it's disrupted, hair follicles can be damaged, but blocking certain pathways can restore hair growth.

The mTurq2-Col4a1 mouse model shows that cells can divide while attached to stable basement membranes during development.

Too much β-catenin activity can mess up the development of mammary glands and make them more like hair follicles.