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Different types of PPARγ are found in varying amounts in human skin and its parts, which could affect how skin treatments work.

A gene called APCDD1, which controls hair growth, is found to be faulty in a type of hair loss called hereditary hypotrichosis simplex.

Activin helps skin growth and healing mainly through stromal cells and affects keratinocytes based on its amount.

Some cells may slow melanoma growth, a protein could affect skin pigmentation, a gene-silencing method might treat hair defects, skin bacteria changes likely result from eczema, and a defensin protein could help treat multiple sclerosis.

DNMT3A is crucial for healthy skin and hair growth.

Notch1 signaling is impaired in hidradenitis suppurativa, affecting skin and hair cells.

New treatments targeting specific genes show promise for treating keratin disorders.

A girl had rickets due to a gene mutation affecting vitamin D response.

Ca_v1.2 affects hair growth and could be a target for hair loss treatments.

Retinoids increase steroid sulfatase activity in leukemia cells through RARα/RXR and involves certain pathways like phosphoinositide 3-kinase and ERK-MAP kinase.

The document explains the genetic causes and characteristics of inherited hair disorders.

Researchers found a genetic link for hereditary hair loss but need more analysis to identify the exact gene.

Blocking the CCR5 receptor may be a new way to treat hair loss from alopecia areata.

A person with a specific gene mutation had extra teeth, unique jaw and hair features not seen before in this condition.

STAT3 signaling is important for healthy skin and hair follicles, and its disruption can lead to skin conditions like atopic dermatitis.

Targeted siRNA therapy may be a promising treatment for KID syndrome by reducing mutant gene expression and improving cell communication.

A mutation in the KRT71 gene causes a hair disorder by disrupting hair follicle structure and texture.

New findings suggest potential treatments for melanoma, hyperpigmentation, hair defects, and multiple sclerosis, and show skin microbiome changes don't cause atopic dermatitis.

BMP2 and BMP7 have opposite roles in feather formation.

Different skin stem cells help heal wounds, with hair follicle cells becoming more important over time.

XEDAR triggers a specific signaling pathway in cells.

Fat cells help recruit healing cells and build skin structure during wound healing.

A mutation in the IKZF1 gene causes immune system overactivity, linked to autoimmune diseases like lupus.

Epidermolysis bullosa simplex causes easily blistered skin due to faulty skin cell proteins, leading to new treatment ideas.

New gene variants linked to a rare inherited hair loss disorder were found in three Chinese families.

Certain cancer drugs interact with skin bacteria to cause acne-like rashes.

Scientists found gene mutations that affect hair loss, skin stem cells, and skin disorders, and identified drugs that may help treat blood vessel and skin conditions.

Hair follicle development is controlled by interactions between skin tissues and specific molecular signals.

Inhibiting AP1 in mice skin causes structural changes and weakens the skin barrier.

The p.P25L mutation in the KRT5 gene causes a rare skin condition that worsens over time and may lead to hair loss starting in young adulthood.