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The conclusion is that genetic changes in the glucocorticoid receptor can lead to conditions affecting stress response, immunity, and metabolism, requiring personalized treatment.

The review suggests thorough evaluation and genetic testing for proper diagnosis and treatment of Chrousos syndrome.

Researchers found a new gene variant linked to a rare bone disease, which doesn't always cause symptoms in carriers.

Researchers found new potential treatments for conditions related to the androgen receptor, like male hormonal contraception, by testing thousands of compounds.

No clear link between androgen receptor variation and hair loss, but more research needed.

Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

OsUEV1B protein is essential for controlling phosphate levels in rice.

Vitamin D receptor interacts with certain dietary components to help prevent diseases and regulate hair growth.

Foxp3 is crucial for regulatory T cell function, and targeting these cells may help treat immune disorders.

5α‐reductase isozymes are crucial for prostate development and health, and targeting them can help prevent and treat prostate issues.

The Foxn1 gene mutation causes hairlessness and immune system issues, and understanding it could lead to hair growth disorder treatments.

Key genes are crucial for Drosophila wing development and could be insecticide targets.

GLI2 increases follistatin production in human skin cells.

Hairless protein is crucial for healthy skin and hair, and its malfunction can cause hair loss.

Researchers found a genetic link for hereditary hair loss but need more analysis to identify the exact gene.

The Hairless gene is crucial for healthy skin and hair growth.

Two new mutations in the AR gene linked to severe androgen insensitivity were found.

Vitamin D receptor is important for regulating hair growth and wound healing in mice.

Mutations in the Vitamin D receptor gene can cause hair loss similar to mutations in the Hairless gene.

Mutations in the FOXN1 gene cause severe immune issues but don't affect hair and nails.

Mutations in the NR3C1 gene cause a rare condition that affects hormone signaling and can lead to various symptoms, with dexamethasone as a treatment option.

CXXC5 is a protein that controls cell growth and healing processes, and changes in its activity can lead to diseases like cancer and hair loss.

Genes and epigenetic changes are important in the development of Polycystic Ovary Syndrome.

Vitamin D receptor binds similarly to natural and synthetic ligands, affecting gene regulation.

Hairless protein can block vitamin D activation in skin cells.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

Two rare genetic diseases cause severe rickets in children due to defects in vitamin D metabolism.

Involucrin gene expression is controlled by specific proteins and signaling pathways.

Genetic defects in the glucocorticoid receptor gene can cause conditions with abnormal sensitivity to stress hormones, and other factors may also affect this sensitivity.

17β-estradiol can reduce inflammation in the skin.