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Stress activates a special function of the Vitamin D receptor with the help of c-Jun, which can also prevent cell death.

Vitamin D-dependent rickets Type II causes bone problems and hair loss, and doesn't improve with Vitamin D treatment.

People with alopecia areata often have lower vitamin D levels, which are linked to more severe and longer-lasting hair loss, but vitamin D receptor levels in the skin don't show the same pattern and don't predict treatment success.

HVDRR is caused by VDR gene mutations, leading to vitamin D resistance, treatable with high calcium doses, but alopecia remains permanent.

Low vitamin D can worsen pediatric alopecia areata.

Removing vitamin D and calcium receptors in mice skin cells slows down skin wound healing.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

Topical vitamin D is useful for some skin conditions but not effective for others, and more research is needed.

Researchers created a mouse model of a type of rickets that does not cause hair loss.

Vitamin D Receptor is needed for hair growth in mice but not for skin stem cell maintenance.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

Vitamin D levels do not affect the risk of developing alopecia areata.

Vitamin D is crucial for bone health and preventing serious diseases.

Vitamin D receptor is important for regulating hair growth and wound healing in mice.

Targeting the Wnt pathway might help treat hair loss.

People with Alopecia areata often have lower Vitamin D levels, which might affect the severity of their condition.

Low vitamin D receptor levels found in hair loss patients; topical vitamin D treatment suggested.

Vitamin D is important for regulating calcium, phosphate, and hair health.

Baldness does not affect vitamin D levels in men.

Patients with myotonic dystrophy often have skin problems that suggest early aging and vitamin D issues, and the severity of these problems is linked to their genetic condition and vitamin D levels.

Vitamin D and calcium are essential for normal hair growth.

Vitamin D boosts a specific protein in skin cells linked to hair follicles.

The vitamin D receptor helps regulate skin and hair health independently of its usual vitamin D ligand.

A genetic mutation caused severe rickets and alopecia in an Indian patient, but high-dose calcium and phosphate treatment improved their condition.

VDUP1 is found in skin and hair follicles, interacts with sciellin, and may help regulate skin cell differentiation.

High Nesfatin-1 and low Vitamin D may increase blood pressure and heart rate in women with PCOS.

Vitamin D boosts a specific gene activity in kidney cells that could improve heart and kidney function.

Vitamin D is important for skin health and can affect various skin diseases.

Vitamin D deficiency is common in women with PCOS and linked to some metabolic problems, but not the main cause of their metabolic issues.

Lack of Vitamin D receptor changes skin structure and increases certain immune cells in the skin.