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The document suggests that certain protein deficiencies and scalp blistering in Epidermolysis Bullosa may cause hair loss.

The mTurq2-Col4a1 mouse model shows how the basement membrane develops in live mammals.

Melanoma's complexity requires personalized treatments due to key genetic mutations and tumor-initiating cells.

Hair loss in autoimmune blistering skin diseases varies and may regrow with disease control.

Collagen XVII is important for skin aging and wound healing.

The document concludes that stem cells and their environments are crucial for skin and hair health and have potential for medical treatments.

People with Down syndrome have a higher risk of skin disorders and need better screening and treatment.

Bovines can have rare inherited skin diseases with specific symptoms like hair loss, fragile skin, and abnormal porphyrin buildup.

Rare ULBP3 gene changes may raise the risk of Alopecia areata, a certain FAS gene deletion could cause a dysfunctional protein in an immune disorder, and having one copy of a specific genetic deletion is okay, but two copies cause sickle cell disease.

Skin development in mammals is controlled by key proteins and signals from underlying cells, involving stem cells for maintenance and repair.

Scientists made a mouse that shows how a specific protein in the skin changes and affects hair growth and shape.

Double-stranded RNA helps regenerate hair follicles by increasing retinoic acid production and signaling.

Hair loss and aging are caused by the breakdown of a key protein in hair stem cells.

The study concludes that mutations in the AEBP1 gene can cause a form of Ehlers-Danlos syndrome and should be considered in diagnosis.

The document concludes that ERBB2 mutations are common in extramammary Paget disease and may respond to systemic treatments like cancer immunotherapy.

Golden oyster mushroom extract may protect skin cells from aging by its antioxidant properties.

Understanding skin structure and development helps diagnose and treat skin disorders.

Alopecia in patients with epidermolysis bullosa varies in severity and is often caused by skin blistering or trauma.

β-catenin is essential for stem cell activation and proliferation in hair follicles.

A WNT10A gene mutation leads to ectodermal dysplasia by disrupting cell growth and differentiation.

Skin lesions in Carney complex are likely caused by a specific group of skin cells that promote pigment production due to a genetic mutation.

Intralesional chemotherapy with 5-fluorouracil and methotrexate may worsen keratoacanthoma-type skin cancer in transplant patients.

Certain genes are linked to the quality of cashmere in goats.

FKBP10 and FBN2 are key proteins for hair growth in cashmere goats.

Epidermolysis bullosa simplex causes easily blistered skin due to faulty skin cell proteins, leading to new treatment ideas.

People with Collagen VI-related myopathies may often have hair loss and scalp issues.

New treatments for skin damage from UV light using stem cells and their secretions show promise for skin repair without major risks.

The Wnt/β-catenin pathway is important for body functions and diseases, and targeting it may treat conditions like cancer, but with safety challenges.

A rare benign skin tumor showed unusual features of sebaceous and sweat glands, important for correct diagnosis.