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Hair follicle-derived IL-7 and IL-15 are crucial for maintaining skin-resident memory T cells and could be targeted for treating skin diseases and lymphoma.

Stress hormones and autoimmune reactions can cause hair loss.

Topical immunotherapy can treat alopecia areata, but its effectiveness varies and the exact mechanism is unclear.

The L412F variant of TLR3 is linked to skin infections, more viral infections, and autoimmune issues.

Dengue virus can infect human hair follicle cells and may cause hair loss.

Immune cells help regulate hair growth, and better understanding this can improve hair loss treatments.

Restoring immune privilege in hair follicles could help treat certain types of hair loss.

Somatostatin may help protect hair follicles from immune attacks.

Researchers found four genes that could help diagnose severe alopecia areata early.

The workshop discussed the role of a protein called calreticulin in health and disease, its potential as a treatment target, and its possible use as a disease marker.

The document concludes that the immune-inhibitory environment of the hair follicle may prevent melanoma development.

Aged individuals heal wounds less effectively due to specific immune cell issues.

Alopecia areata is likely an autoimmune disease with unclear triggers, involving various immune cells and molecules, and currently has no cure.

New protein changes may be involved in the immune attack on hair follicles in alopecia areata.

The current understanding of frontal fibrosing alopecia involves immune, genetic, hormonal factors, and possibly environmental triggers, but more research is needed for effective treatments.

The nail matrix has a reduced immune response, protecting it from autoimmunity.

Hair follicles prevent NK cell attacks to avoid hair loss.

Stress may trigger hair loss by affecting immune protection in hair follicles.

Targeting the protein Caveolin-1 might help treat a type of scarring hair loss called Frontal Fibrosing Alopecia.

The conclusion is that different types of hair loss in dogs and cats can be cosmetic or serious, and affected animals should not be bred.

ILC1-like cells can cause alopecia areata by attacking hair follicles.

A specific enzyme is essential for proper hair follicle stem cell development and healthy skin.

Skin bacteria, specifically Staphylococcus aureus, help in wound healing and hair growth by using IL-1β signaling. Using antibiotics on skin wounds can slow down this natural healing process.

The review suggests that other immune cells besides CD8+ T cells may contribute to alopecia areata and that targeting regulatory cell defects could improve treatment.

Scientists are still unsure what triggers the immune system to attack hair follicles in Alopecia areata.

NK cells play a role in skin diseases like eczema and psoriasis.

T-cells in alopecia areata scalp show abnormal regulation, leading to less inflammation.

Self-amplifying RNA could be a better option for protein replacement therapy with lower doses and lasting effects, but delivering it into cells is still challenging.

Recombinant cytokine therapy can cause skin reactions ranging from mild to severe.

The conditions alopecia areata, primary sclerosing cholangitis, and ulcerative colitis may be linked by shared autoimmune and cell death mechanisms.