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After menopause, some women lose scalp hair and gain facial hair, with patterns suggesting different underlying causes.

Hair growth is influenced by hormones and goes through different phases; androgens can both promote and inhibit hair growth depending on the body area.

Antiandrogens are the main treatment for hirsutism, with individualized care and safe, affordable options needed.

Hirsutism in women is often due to hormone sensitivity and has significant psychological effects.

Hirsutism causes significant psychological distress in women.

A gene called BMAL1 plays a role in controlling hair growth.

Testosterone treatment can improve sexual function and bone density in women but may have adverse effects and requires more research on safety and guidelines.

Men with male pattern baldness have a higher risk of aggressive prostate cancer and benign prostatic hyperplasia.

Men's hair loss is not linked to their sex hormone levels, except for lower DHEAS in those with hair loss.

Hirsutism, excessive hair growth in women, is often caused by PCOS and can be managed with medication and personalized treatment plans.

Hormonal treatments like birth control pills can improve acne in women with excess hair growth and hormone imbalances.

Male hormones can cause hair loss, but treatments like Minoxidil and Finasteride can help, and targeting TGF-B1 could be a future solution.

The enzyme 5α-reductase type 1 is important for blood vessel development and fertility in the uterus.

Male yak hair growth is influenced by DHT synthesis, which is promoted by 5α-red1 and AR during growth phases, while E2 may inhibit growth through ERα.

Hormones significantly affect women's sexual function, and more research is needed to improve treatments for sexual dysfunction with minimal side effects.

Testosterone can both promote hair growth and cause baldness by affecting hair growth signals.

The review suggests a comprehensive approach to treat hirsutism, focusing on hair removal, medication, and managing emotional effects.

Clinicians should use social and prescription data to track trends in performance-enhancing drug use.

Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

Cell transplantation faces challenges in genitourinary reconstruction, but alternative tissue sources and microencapsulation show promise.

Male androgenetic alopecia involves hair follicle miniaturization due to DHT, with potential treatments using inhibitors and blockers.

PCOS may increase the risk of certain cancers.

The enzyme 5α-reductase is important for proper blood vessel development during the fertility-related transformation of the uterus lining.

New factors in female hair loss include genetics, hormones, stress, and inflammation; future treatments should also focus on these areas and consider the patient's emotional well-being.

A specific DNA sequence caused hair loss in male mice by activating immune cells and increasing a certain immune signal.

Flutamide caused damage to male rat reproductive organs and may affect fertility.

The document concludes that early diagnosis and treatment of PCOS in teenagers is important for managing symptoms and preventing long-term health problems.

The 2004 hair research meeting presented new findings on hair cell differentiation, genetic factors in hair loss, hair pigmentation, and potential targeted therapies.

The workshop highlighted the genetic links and psychological impacts of hair loss and skin disorders.

Women with androgenetic alopecia (hair loss) have normal prostate specific antigen levels but higher testosterone levels.