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Mutations in the Vitamin D receptor gene can cause hair loss similar to mutations in the Hairless gene.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

Vitamin D3 is important for hair growth and protecting skin from damage caused by long-term sun exposure.

VDR regulation varies by tissue and is crucial for its biological functions.

Two siblings with a genetic mutation had a form of rickets that doesn't respond to vitamin D.

High CRP levels could indicate vitamin D deficiency in people with alopecia areata.

An elderly man's hair grew back after a treatment that transferred healthy gut bacteria.

Vitamin D and calcium are important for skin stem cell function and wound healing.

Mixing calcipotriol with a steroid is almost as effective as using a steroid alone for alopecia areata and has fewer side effects.

Vitamin D might help treat some types of hair loss, but more research is needed.

Kids with alopecia areata had lower vitamin D levels than healthy kids.

Combination pharmacotherapy is generally more effective for treating keloids and hypertrophic scars.

Low vitamin D levels are common in people with Alopecia Areata.

A Chinese family had a child with a specific gene mutation causing vitamin D-resistant rickets, but the child improved with calcium and low-dose calcitriol.

People with Alopecia Areata often have lower vitamin D levels, and vitamin D supplements might help treat it.

The hair test for vitamin D could be a useful alternative to blood tests, providing a longer-term vitamin D status, but more research is needed.

Topical vitamin D is useful for some skin conditions but not effective for others, and more research is needed.

Some growth factors, while important for normal body functions, can cause diseases when not regulated properly.

Melasma is a skin condition linked to female hormones, genetics, UV exposure, and certain medications, but not to pituitary, adrenal, or thyroid diseases.

No treatment fully prevents hair loss from chemotherapy yet.

Removing REDD1 in mice increases skin fat by making fat cells larger and more numerous.

DPP4-positive fibroblasts play a major role in producing proteins that lead to skin fibrosis.

Fractional photothermolysis helps wounds heal with minimal scarring.

BRG1 is essential for skin cells to move and heal wounds properly.

Hair regrowth was seen in 83% of children with alopecia, immune system plays a role in the condition, and various treatments showed effectiveness for hair and nail disorders.

Skin's Regulatory T cells are crucial for maintaining skin health and could be targeted to treat immune-related skin diseases and cancer.

An imbalance between certain immune cells is linked to a chronic skin condition and may be influenced by obesity, smoking, and autoimmune issues.

TGF-β2 plays a key role in human hair growth and development.

Scalp cooling is the most effective FDA-approved method to prevent chemotherapy-induced hair loss, but more research is needed for other treatments.

Understanding how cells die in the skin is important for treating skin diseases and preventing hair loss.