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Increasing the levels of a protein called FGF9 can promote hair growth, but humans may not respond the same way due to a lack of certain cells.

Certain types of T cells are essential for healthy skin and play a role in skin diseases, but more research is needed to improve treatments.

γδ T cells help with hair growth during wound healing in mice.

A protein from certain immune cells is key for new hair growth after skin injury in mice.

Regulatory T cells are essential for normal and improved wound healing in mice.

Gamma delta T cells in the skin help with healing and defense but can also cause autoimmune issues, and more research is needed to understand how they are activated.

T cells and bacteria in the gut and skin help maintain health and protect against disease.

Thymic transplantation normalized some T-cells but not others, maintaining immune function.

Inflammation plays a key role in activating skin stem cells for hair growth and wound healing, but more research is needed to understand how it directs cell behavior.

T-cell reconstitution after thymus transplantation can cause hair whitening and loss.

New treatments targeting T-cell pathways are needed for better alopecia areata management.

Imiquimod cream activates hair follicle stem cells and causes early hair growth by changing immune cells and certain protein expressions.

Mice can grow new hair follicles after skin wounds through a process not involving existing hair stem cells, but requiring more research to understand fully.

New hair can grow from large wounds in mice, but less so as they age, involving reprogramming of skin cells and specific molecular pathways.

Small molecule DMF improves psoriasis and multiple sclerosis, adult skin cells can be made to grow new hair, certain skin cells initiate hair growth, IL-17C controls gut health and can cause skin inflammation, and skin cells produce IL-17 that can lead to psoriasis.

Keratinocytes help heal skin wounds by interacting with immune cells and producing substances that kill pathogens.

A protein called FGF9 helps regenerate hair follicles in mice after skin damage, and increasing FGF9 could potentially help human hair growth.

Hair growth phase and certain genes can speed up wound healing, while an inflammatory mediator can slow down new hair growth after a wound. Understanding these factors can improve tissue regeneration during wound healing.

Stress can cause hair loss by negatively affecting hair follicles and this effect might be reversed with specific treatments.

The skin has a complex immune system that is essential for protection and healing, requiring more research for better wound treatment.

Human prenatal skin develops an immune network early on that helps with skin formation and healing without scarring.

Hair can regrow in large wounds through a process similar to how hair forms in embryos, and understanding this could lead to new treatments for hair loss or scarring.

The skin and thymus develop similarly to protect and support immunity.

Newborn screening and gene therapy are expected to improve outcomes for Omenn syndrome patients.

The conclusion is that Treg-targeted therapies have potential, but more knowledge of Treg biology is needed for effective treatments, including for cancer.

Hair follicles can help wounds heal faster and this knowledge could be used to treat chronic skin ulcers, with a potential use of a special stem cell hydrogel to enhance healing.

Pantethine may boost the immune system's ability to fight sarcoma.

Understanding different types of skin cells, especially fibroblasts, can lead to better treatments for wound healing and less scarring.

Blood-derived CD34+ cells speed up healing, reduce scarring, and regrow hair in skin wounds.

Gene network oscillations inside hair stem cells are key for hair growth regulation and could help treat hair loss.