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The document concludes that mouse models are crucial for studying hair biology and that all mutant mice may have hair growth abnormalities that require detailed analysis to identify.

Hormonal treatment is effective for women with acne not helped by usual treatments, especially if they have hormonal imbalances.

Women with female pattern hair loss have lower Vitamin D3 levels.

Estrogens significantly influence hair growth by interacting with receptors in hair follicles and may help regulate the hair growth cycle.

Turning white fat into brown-like fat could help fight obesity and type 2 diabetes.

Female Pattern Hair Loss affects women's self-esteem and needs more research for better treatment.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

A boy with alopecia regrew hair using a vitamin D cream after other treatments failed.

Taking vitamin D might improve life for MS patients and reduce skin side effects from alemtuzumab treatment.

Mutations in the androgen receptor gene cause Androgen Insensitivity Syndrome, affecting sexual development.

Certain gene patterns in breast cancer are linked to how active hormone receptors are and could affect patient survival.

Changes in testosterone and estrogen receptor genes can affect how men age, influencing body fat, hair patterns, and possibly leading to skin disorders.

The length of the CAG repeat in the androgen receptor gene affects the risk and progression of prostate cancer, BPH, infertility, and undermasculinized genitalia.

DHT affects bone growth by altering gene activity in osteoblasts, potentially complicating steroid use.

Hair follicles are essential for skin health, aiding in hair growth, wound healing, and immune function.

Thyroid hormone receptors are essential for hair growth and wound healing.

Mutations in the Vitamin D receptor gene can cause hair loss similar to mutations in the Hairless gene.

A specific gene mutation causes vitamin D-resistant rickets and hair loss.

A new gene mutation causes vitamin D-resistant rickets and hair loss in two siblings.

A genetic mutation caused severe rickets and alopecia in an Indian patient, but high-dose calcium and phosphate treatment improved their condition.

A girl had rickets due to a gene mutation affecting vitamin D response.

Most Hairless gene mutations reduce its ability to work with the Vitamin D Receptor, which might explain a certain type of hair loss.

The vitamin D receptor is crucial for bone health and affects various body systems, with mutations potentially leading to disease.

The study found that certain mutations in the vitamin D receptor can cause rickets and potentially affect hair growth.

Gene therapy with a vitamin D receptor gene improved hair growth in rats with a type of rickets-related baldness.

Hairless protein can block vitamin D activation in skin cells.

LEF1 interacts with Vitamin D Receptor, affecting hair follicle regeneration and this could be linked to hair loss conditions.

Vitamin D boosts a specific gene activity in kidney cells that could improve heart and kidney function.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

Some people have genetic mutations that make them resistant to vitamin D, leading to rickets even with enough vitamin D intake.