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Tunisian children with hereditary vitamin D-resistant rickets showed improvement with calcium treatment, and new genetic mutations were identified.

Two siblings with a rare type of rickets and hair loss had low vitamin D levels, which is not typical for their condition.

A child with a rare type of rickets showed some improvement with high doses of vitamin D, but such conditions often respond poorly to treatment.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requiring high doses of calcium and vitamin D for treatment.

Two rare genetic diseases cause severe rickets in children due to defects in vitamin D metabolism.

Vitamin D is important for skin health and can affect various skin diseases.

A mutation in the hairless gene speeds up severe itchy skin in mice on a special diet.

Hairless protein is crucial for healthy skin and hair, and its malfunction can cause hair loss.

The Hairless gene is crucial for healthy skin and hair growth.

Researchers created a mouse model of a type of rickets that does not cause hair loss.

The HR protein's role as a repressor is essential for controlling hair growth.

Combining skin tissue pathology with genetics has greatly improved the diagnosis and understanding of certain skin diseases.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

Vitamin D receptor helps control hair growth genes in skin cells.

Mice without the vitamin D receptor have bone issues and other health problems, suggesting vitamin D is important for preventing various diseases in humans.

Removing part of the vitamin D receptor stops vitamin D from working properly.

Vitamin D receptor interacts with certain dietary components to help prevent diseases and regulate hair growth.

Lampreys have a functional vitamin D receptor that may help detoxify harmful substances.

Vitamin D receptor is crucial for starting hair growth after birth.

Vitamin D Receptor is needed for hair growth in mice but not for skin stem cell maintenance.

Vitamin D receptor may help protect against UV-induced skin cancer.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

Vitamin D receptor helps control hair growth and could be used to treat certain skin tumors.

Vitamin D and its receptor may help prevent skin cancer.

Vitamin D receptor is important for regulating hair growth and wound healing in mice.

The vitamin D receptor helps regulate skin and hair health independently of its usual vitamin D ligand.

Vitamin D Receptor is crucial for normal skin and hair growth.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.