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A new PAX9 gene mutation causes missing teeth and hair problems, but not skin or nail issues.

Researchers found a new mutation in the EDA gene that likely causes missing teeth and mild skin symptoms in one family.

Mutations in the WNT10A gene can cause skin, hair, teeth, and other disorders, and may also affect other areas like kidney and cancer, with potential for targeted treatments.

Different fish use the same genes to regrow teeth.

WNT10A mutations often cause ectodermal dysplasias, with males showing more tooth issues than females.

Different species use the same genes for tooth regeneration.

Msx and Dlx genes are crucial for development, controlling cell behaviors like growth and differentiation through their roles as gene regulators.

A woman with a rare hormone resistance condition also had missing teeth and hair loss, which might be new symptoms of her genetic disorder.

High TSPEAR levels in colorectal cancer predict worse outcomes.

High TSPEAR levels in colorectal cancer predict worse outcomes.

Regenerated fully functional hair follicles using stem cells, with potential for hair regrowth therapy.

Hair, teeth, and mammary glands develop similarly at first but use different genes later.

A WNT10A gene mutation leads to ectodermal dysplasia by disrupting cell growth and differentiation.

Scientists successfully created and transplanted bioengineered hair follicles that function like natural ones, suggesting a new treatment for hair loss.

The WNT signaling pathway is important in many diseases and targeting it could offer new treatments.

The article explains the genetic causes and symptoms of various hair disorders and highlights the need for more research to find treatments.

Boys with less severe EDA mutations in XLHED have milder symptoms and better sweat and hair production.

Researchers found genetic mutations causing hypohidrotic ectodermal dysplasia in 88% of studied patients and identified new mutations and genetic variations affecting the disease.

Future research on ectodysplasin should explore its role in diseases, stem cells, and evolution, and continue developing treatments for genetic disorders like hypohidrotic ectodermal dysplasia.

Proper control of β-catenin activity is crucial for development and preventing diseases like cancer.

MendelVar is a tool that helps identify important genes by combining GWAS data with Mendelian disease information.

Scientists now better understand the genetics of hypohidrotic ectodermal dysplasia, leading to more accurate diagnoses and potential new treatments.

The document is a detailed medical reference on skin and genetic disorders.

WNT10B is linked to cancer development and affects survival and disease progression in various cancers.

Certain genetic changes in the LSS gene cause a rare skin and hair condition.

A new EDA gene mutation was found in a Chinese family with a specific skin disorder.

The Wnt/β-catenin pathway is important for body functions and diseases, and targeting it may treat conditions like cancer, but with safety challenges.

Ectodysplasin signaling is crucial for skin appendage development, requiring specific doses and durations.

Rare ULBP3 gene changes may raise the risk of Alopecia areata, a certain FAS gene deletion could cause a dysfunctional protein in an immune disorder, and having one copy of a specific genetic deletion is okay, but two copies cause sickle cell disease.

Cactus extract from Notocactus ottonis may help promote hair growth.