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The cause of alopecia areata is likely a mix of genetics, immune system issues, and environmental factors, with more research needed to understand it fully.

Deleting MED1 in skin cells causes hair loss and skin changes.

Hair follicle samples effectively show how well the drug MK-0752 targets and engages with the Notch pathway.

People with atopic dermatitis have more CD4+ T cells that respond to a certain bacterial lipid, which may play a role in the skin condition's inflammation.

Testosterone can both promote hair growth and cause baldness by affecting hair growth signals.

Human hair follicle stem cells can turn into multiple cell types but lose some of this ability after being grown in the lab for a long time.

Hair follicle-derived IL-7 and IL-15 are crucial for maintaining skin-resident memory T cells and could be targeted for treating skin diseases and lymphoma.

Skin cells release substances important for healing and fighting infection, and understanding these could improve skin disorder treatments.

Immune cells affect hair growth and could lead to new hair loss treatments.

RANKL improves the immune response against herpes simplex virus by enhancing T cell activation and could help develop better treatments or vaccines.

Gene network oscillations inside hair stem cells are key for hair growth regulation and could help treat hair loss.

Interferon gamma alone can't cause alopecia areata in C3H/HeJ mice.

All-trans retinoic acid causes hair loss by increasing TGF-β2 in hair follicle cells.

Alopecia areata is likely caused by a combination of genetic factors and immune system dysfunction, and may represent different diseases with various causes.

Certain immune cells are linked to non-scarring hair loss, suggesting potential for immune-targeted treatments.

Low-Level Laser Therapy can stimulate healing and cell function, potentially leading to wider medical use.

The Rotterdam Study updated findings on elderly health, focusing on heart disease, genetics, lifestyle effects, and disease understanding.

Y27632 increases cell growth through EGFR signaling, not ROCK1/2.

Fat cells are important for healthy skin, hair growth, and healing, and changes in these cells can affect skin conditions and aging.

Researchers created rat liver stem cells that could help repair liver failure in rats and may be useful for studying human liver diseases.

Kennedy's disease leads to muscle weakness and sensory issues, has no cure but manageable symptoms, and future treatments look promising.

Blocking certain immune signals can reduce skin damage from radiation therapy.

Trichloroethylene causes skin inflammation in mice by increasing certain immune proteins.

Future research on ectodysplasin should explore its role in diseases, stem cells, and evolution, and continue developing treatments for genetic disorders like hypohidrotic ectodermal dysplasia.

Mice lacking the PPARγ gene in their fat cells had almost no fat tissue, severe metabolic problems, and abnormal development of other fat-related tissues.

Genetic mutations can cause hair growth disorders by affecting key genes and signaling pathways.

Fat cells help recruit healing cells and build skin structure during wound healing.

ILC1-like cells can independently cause alopecia areata by affecting hair follicles.

Alopecia areata is an autoimmune disease with genetic links, treatable with certain medications, and can affect mental health.

Using fat-derived stem cells with the drug meglumine antimoniate can help control skin disease and reduce parasites in mice with leishmaniasis.