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Alopecia areata is an autoimmune disease causing patchy hair loss, often with other autoimmune disorders, but its exact causes are unknown.

Targeting the IL-23/IL-17 pathway effectively treats several inflammatory skin diseases.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

Foxp3 is crucial for regulatory T cell function, and targeting these cells may help treat immune disorders.

Newborn mouse skin cells can grow hair and this process can be recreated in adult cells to potentially help with hair loss.

Lichen planopilaris is a chronic, scarring hair loss condition with no definitive cure, requiring accurate diagnosis and treatment to manage symptoms.

Animal models have helped understand hair loss from alopecia areata and find new treatments.

Older skin has higher cancer risk due to inflammation and stem cell issues.

Special immune cells called Regulatory T cells help control skin inflammation and repair in various skin diseases.

Type I interferons play a key role in the development of various skin diseases.

PI3K/Akt pathway is crucial for hair growth and regeneration.

The document concludes that certain mutations may contribute to the inflammation in hidradenitis suppurativa and suggests that targeting TNFα could be a treatment strategy.

Hair follicle bulge cells don't help skin regrow after glucocorticoid damage; interfollicular epidermis cells do.

Double-stranded RNA causes inflammation in hair follicle cells, which may help understand and treat alopecia areata.

The composition with carnitine, thioctic acid, and saw palmetto extract may effectively reduce inflammation in hair follicle cells.

COVID-19 might worsen symptoms and progression of benign prostatic hyperplasia, possibly due to inflammation and metabolic disturbances in the prostate gland. More research is needed to confirm this.

CBD can either reduce inflammation and not affect hair growth at low levels or inhibit hair growth at high levels.

Cardiovascular drugs can cause various skin problems, so recognizing these reactions is important.

Newborns with the common rash Erythema Toxicum have many active mast cells in their skin, but these cells don't produce the LL-37 peptide.

Finasteride raises suicide-linked aggression and stops clozapine's positive effects in schizophrenia animals.

Human prenatal skin develops an immune network early on that helps with skin formation and healing without scarring.

Both topical calcipotriol and narrowband UVB improved alopecia, but combining them didn't enhance the effect on hair loss severity, despite higher vitamin D3 levels.

Key genes linked to hair growth and cancer were identified in hairless mice.

Some thymic peptides can increase human hair growth, while others may inhibit it.

Chicken feather gene mutation helps understand human hair disorders.

Ginsenoside Rg1 protects mouse skin from UVB damage and helps control inflammation.

TLR2 is important for hair growth and can be targeted to treat hair loss.

Blocking Prostaglandin D₂ may help treat hair loss.

Certain genes influence immunoglobulin levels in Chinese Holstein cows, which can improve calf health.

Blocking Oncostatin M's role in the JAK-STAT pathway can stimulate hair growth in mice.