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Vitamin D receptor is crucial for starting hair growth after birth.

SARMs may be an effective treatment for a certain type of breast cancer by blocking cancer growth and spread.

Vitamin D and its receptor may help prevent skin cancer.

Parathyroid hormone-related protein helps control hair growth phases in mice.

Vitamin D receptor may help protect against UV-induced skin cancer.

A specific gene mutation causes vitamin D-resistant rickets and hair loss.

The vitamin D receptor is essential for skin stem cells to grow, move, and become different cell types needed for skin healing.

CD8+ T cells are involved in alopecia areata and may cause disease relapse.

Disrupting Acvr1b in mice causes severe hair loss and thicker skin.

Most Hairless gene mutations reduce its ability to work with the Vitamin D Receptor, which might explain a certain type of hair loss.

RXR-alpha is strongly expressed in both normal and psoriatic skin and may help in skin cell differentiation and hair growth.

Skin problems like acne, dry skin, and nail and hair changes are common in patients taking EGFR inhibitors.

Selective androgen receptor modulators (SARMs) are a promising type of drug for various health conditions due to their targeted actions.

LEF1 interacts with Vitamin D Receptor, affecting hair follicle regeneration and this could be linked to hair loss conditions.

Mice skin has components that could help with hair growth and might be used for diabetes treatment.

The review found no clear link between vitamin D receptor gene variations and polycystic ovary syndrome.

The vitamin D receptor can work without its usual activating molecule.

ROOT HAIR SPECIFIC 10 (RHS10) reduces the length of root hairs in Arabidopsis plants.

Pioglitazone usually doesn't effectively treat or cure lichen planopilaris.

PTHrP and its receptor can control blood vessel growth and hair development in mouse skin.

Stress activates a special function of the Vitamin D receptor with the help of c-Jun, which can also prevent cell death.

People with alopecia areata often have lower vitamin D levels, which are linked to more severe and longer-lasting hair loss, but vitamin D receptor levels in the skin don't show the same pattern and don't predict treatment success.

Removing vitamin D and calcium receptors in mice skin cells slows down skin wound healing.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

Blocking Wnt/β-catenin signaling with EGF receptor is necessary for proper hair growth.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

Blocking a specific receptor slows down hair loss in mice.

VEGF stimulates hair cell growth and increases growth receptor levels through a specific signaling pathway.

The guidelines help doctors manage skin problems from certain cancer treatments to improve patients' lives.

Vitamin D receptor is important for regulating hair growth and wound healing in mice.