Search

everythinglearnresearchcommunity

for

Search:↓

Using travoprost for glaucoma may cause extra hair growth and darker skin around the eyes, but these effects can reverse after stopping the drug.

Latanoprost eye drops caused excessive hair growth and eyelash whitening in a woman.

Latanoprost eye drops caused excessive cheek hair growth and eyelash whitening in a woman.

LPA3 is crucial for embryo implantation and links LPA to prostaglandin signaling.

A woman kept her eyelashes during chemotherapy possibly because of her glaucoma eye drops.

Blocking Oncostatin M's role in the JAK-STAT pathway can stimulate hair growth in mice.

Double-stranded RNA activates a pathway that causes a skin protein to be expressed in the wrong place.

PGF_2α receptors in human eyelids are found in specific parts of hair follicles, explaining eyelash changes with glaucoma treatment.

Travoprost can help eyelash growth but may cause darkening around the eyes.

Combining platelet-rich plasma therapy with prostaglandin-F eye drops can significantly regrow hair in alopecia universalis.

Topical latanoprost and bimatoprost ophthalmic solutions don't help eyelash growth in patients with alopecia areata.

Stopping minoxidil and using prostaglandin analogues improved the man's eye condition.

Some treatments, like corticosteroids and sensitizing agents, can help with alopecia areata, but more high-quality research is needed.

Two children grew extra hair from taking omeprazole, which went away after they stopped the medication.

Wnt3a activates certain genes in hair follicle cells, including a newly discovered one, EP2, which may affect hair growth.

People with androgenetic alopecia have different lipid levels in their blood, which vary between men and women, and may be linked to a higher risk of metabolic syndrome.

Prostaglandin D2 increases testosterone production in skin cells through a process involving reactive oxygen species, which could be a new target for treating hair loss and other skin conditions driven by testosterone.

The symposium concluded that a specific lipid might cause hair loss, inflammation is a key factor, and stem cells in bald scalps aren't working right, suggesting new treatment targets.

The document concludes that there are various treatments for different types of alopecia, but more research is needed for evidence-based treatments.

Hair loss can be treated with common methods like minoxidil and finasteride, but new potential treatments include growth factors, cytokines, and platelet-rich plasma injections.

Possible new treatments for common hair loss include drugs, stem cells, and improved transplants.

The enzyme sEH is important for hair growth and its inhibition could help treat hair loss.

Alopecia areata can be managed with various treatments, and severe cases should be referred to dermatologists.

Cyclooxygenase-2 overexpression in mice skin causes hair loss like human androgenetic alopecia.

Some nonsurgical hair loss treatments are effective, especially if started early.

The research explains how a human enzyme binds and processes its substrate, which could relate to its role in biological functions and hair loss.

Some alternative vitiligo treatments show promise, but none are as effective as psoralens and UVA.

Certain immune cells are more common on the top of the head and might help predict or treat common hair loss.

Human hair follicles can make and process prostaglandins, which may affect hair growth.

Blocking Prostaglandin D₂ (PGD₂) could help treat hair loss.